ライフサイエンスコーパス: herpes simplex

1 Herpes simplex encephalitis (HSE) is the most common for

2 The herpes simplex encephalitis (HSE) model was used for ind

3 cent reviews; these include the relevance of herpes simplex encephalitis and of epilepsy to AD, the a

4 Although herpes simplex encephalitis has been extensively studied

5 netic association between TRIF mutations and herpes simplex encephalitis in patients.

6 l intervention is crucial to the survival of herpes simplex encephalitis patients; however, many surv

7 al keratitis, highly debilitating and lethal herpes simplex encephalitis, and generalized infections

8 including necrotizing stromal keratitis and herpes simplex encephalitis.

9 Many viruses, including herpes simplex (HSV), are recruited to their host cells

10 ), polyoma (BK) viremia (8.2% vs 11.1%), and herpes simplex infections (5.5% vs 4.0%).

11 Genital herpes simplex is one of the most prevalent sexually tra

12 ree patients with a unilateral and relapsing herpes simplex keratitis (HSK group) that was quiescent

13 ity of life (QoL) in patients with quiescent herpes simplex keratitis compared with control patients

14 group initially were misdiagnosed as having herpes simplex keratitis versus 59 (41.8%) patients who

15 al patients were diagnosed sequentially with herpes simplex keratitis, then Acanthamoeba keratitis be

16 for dendritiform keratopathy, such as prior herpes simplex keratitis, varicella-zoster viral keratit

17 ated with antiviral medications for presumed herpes simplex keratitis; 4 patients underwent diagnosti

18 petic anterior uveitis (AU), owing to either herpes simplex or varicella zoster virus, by using the S

19 lopment of a safe and effective asymptomatic herpes simplex vaccine that is selectively based on CD8(

20 lopment of a safe and effective T-cell-based herpes simplex vaccine.

21 polymerase chain reaction (PCR) analysis for Herpes simplex, varicella zoster, cytomegalovirus, Epste

22 this assessment include adenovirus 8 and 19, herpes simplex virus (HSV) 1 and 2, human immunodeficien

23 Herpes simplex virus (HSV) 1 stimulates type I IFN expre

24 dy of neurotropic viruses in vitroIMPORTANCE Herpes simplex virus (HSV) affects millions of people wo

25 tions that US9 is neuron specific.IMPORTANCE Herpes simplex virus (HSV) and other alphaherpesviruses,

26 Herpes simplex virus (HSV) anterograde transport in neur

27 a broad antimicrobial factor that restricts herpes simplex virus (HSV) by activating type I interfer

28 The herpes simplex virus (HSV) capsid is released into the c

29 his study employed a novel strategy in which herpes simplex virus (HSV) carrying a small interfering

30 Herpes simplex virus (HSV) causes acute and relapsing sy

31 fluid (CSF) commonly predicts the absence of herpes simplex virus (HSV) central nervous system (CNS)

32 After entry into the nucleus, herpes simplex virus (HSV) DNA is coated with repressive

33 During lytic infection, herpes simplex virus (HSV) DNA is replicated by a mechan

34 Herpes simplex virus (HSV) entry into a subset of cells

35 erpesvirus entry mediator (HVEM) facilitates herpes simplex virus (HSV) entry through interactions wi

36 Herpes simplex virus (HSV) establishes a latent reservoi

37 Herpes simplex virus (HSV) establishes latency and react

38 xpression of 'pore dead' GluA1 subunits (via herpes simplex virus (HSV) GluA1-Q582E) in the lateral c

39 Pritelivir is a well-tolerated novel herpes simplex virus (HSV) helicase-primase inhibitor th

40 is known as a transcriptional coactivator of herpes simplex virus (HSV) immediate early (IE) transcri

41 The herpes simplex virus (HSV) infected cell culture polypep

42 Neuron-virus interactions that occur during herpes simplex virus (HSV) infection are not fully under

43 Importance: Genital herpes simplex virus (HSV) infection is a prevalent sexu

44 Herpes simplex virus (HSV) infection is restricted to ep

45 Herpes simplex virus (HSV) infection is widespread in th

46 for antithrombin III and plays a key role in herpes simplex virus (HSV) infection.

47 Studies of herpes simplex virus (HSV) infections of humans are limi

48 Herpes simplex virus (HSV) infections of the central ner

49 During lytic herpes simplex virus (HSV) infections, the virion host s

50 The terminase of herpes simplex virus (HSV) is composed of three subunits

51 Herpes simplex virus (HSV) is investigated not only as a

52 pediatric penetrating keratoplasty (PPK) for herpes simplex virus (HSV) keratitis.

53 ctivator HCF-1 is required for initiation of herpes simplex virus (HSV) lytic infection and for react

54 f aqueous or vitreous humor was positive for herpes simplex virus (HSV) or varicella zoster virus (VZ

55 od to visualize pseudorabies virus (PRV) and herpes simplex virus (HSV) particles in living cells.

56 Despite frequent herpes simplex virus (HSV) reactivation, peripheral nerv

57 The rate of oral herpes simplex virus (HSV) shedding was lower overall, a

58 Chronic infections with herpes simplex virus (HSV) type 1 are highly prevalent i

59 The herpes simplex virus (HSV) type I alkaline nuclease, UL1

60 Samples were tested for herpes simplex virus (HSV) types 1 and 2, Epstein-Barr v

61 synthesis during multiround transmission of herpes simplex virus (HSV) using pulse-labeling with eth

62 e by microinjecting before training a single herpes simplex virus (HSV) vector expressing either CRIS

63 tential role of a TLR4/MCP-1 signal, we used Herpes Simplex Virus (HSV) vectors (amplicons) that reta

64 Presence of CMV, EBV, and herpes simplex virus (HSV) were independent predictors o

65 Caused by the herpes simplex virus (HSV), herpes is a viral infection

66 Infection is caused by 2 subtypes of the herpes simplex virus (HSV), HSV-1 and HSV-2.

67 itro nanomolar irreversible activity against herpes simplex virus (HSV), human papilloma virus, respi

68 cell entry and subsequent lateral spread of herpes simplex virus (HSV), requires the four envelope g

69 Herpes simplex virus (HSV), which triggers the STING sig

70 Herpes simplex virus (HSV)-1 and HSV-2 are significant h

71 cally significant species of simplexviruses, herpes simplex virus (HSV)-1 and HSV-2, with estimated d

72 ACASI), before test HIV counselling, HIV and herpes simplex virus (HSV)-2 testing, and post-test coun

73 In herpes simplex virus (HSV)-infected cells, ND10 bodies a

74 creased) particle-to-PFU ratio in vitro than herpes simplex virus (HSV).

75 enchymal brain inflammation, commonly due to herpes simplex virus (HSV).

76 interferon (IFN) is important for control of herpes simplex virus (HSV-1) in the central nervous syst

77 e delivered to the same sensory neurons that herpes simplex virus (HSV-1) infects when applied periph

78 alone mediates recognition and clearance of herpes simplex virus (HSV1)-infected cells.

79 ablation on tga20/CD11b-thymidine kinase of Herpes simplex virus (HSVTK) cerebellar organotypic cult

80 Oncolytic herpes simplex virus (oHSV) selectively replicates in ca

81 nd tested the fate and efficacy of oncolytic herpes simplex virus (oHSV)-armed mesenchymal stem cells

82 n of foreign DNA-sensing pathways.IMPORTANCE Herpes simplex virus 1 (HSV-1) afflicts 80% of the popul

83 Herpes simplex virus 1 (HSV-1) and HSV-2 are large, doub

84 qualitative detection and differentiation of herpes simplex virus 1 (HSV-1) and HSV-2 DNA in 1,351 cu

85 Herpes simplex virus 1 (HSV-1) and HSV-2 infect many hum

86 f UL37 homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and pseudorabies virus (P

87 hat gB homologs from two alphaherpesviruses, herpes simplex virus 1 (HSV-1) and saimiriine herpesviru

88 te aspects of intrinsic immunity to restrict herpes simplex virus 1 (HSV-1) as well as other viruses.

89 The herpes simplex virus 1 (HSV-1) capsid is a huge assembly

90 ar disease in virus-infected mice.IMPORTANCE Herpes simplex virus 1 (HSV-1) causes cold sores and neo

91 Herpes simplex virus 1 (HSV-1) encodes the multifunction

92 Herpes simplex virus 1 (HSV-1) enters mice via olfactory

93 ponent of cell membranes and is required for herpes simplex virus 1 (HSV-1) entry (1-3).

94 The dissection of the herpes simplex virus 1 (HSV-1) entry mechanism is compli

95 ined whether Cbl mediates the removal of the herpes simplex virus 1 (HSV-1) entry receptor Nectin-1 f

96 Herpes simplex virus 1 (HSV-1) establishes latency withi

97 Herpes simplex virus 1 (HSV-1) establishes lifelong infe

98 ultaneous presence of a helper virus such as herpes simplex virus 1 (HSV-1) for productive replicatio

99 plication greatly enhances expression of the herpes simplex virus 1 (HSV-1) gamma2 late genes by stil

100 Herpes simplex virus 1 (HSV-1) glycoprotein B (gB)-speci

101 gp120) construct fused to a small portion of herpes simplex virus 1 (HSV-1) glycoprotein D (gD) so th

102 The herpes simplex virus 1 (HSV-1) ICP0 protein is an E3 ubi

103 mia nuclear bodies (PML NBs), is a target of herpes simplex virus 1 (HSV-1) ICP0-mediated degradation

104 For example, herpes simplex virus 1 (HSV-1) immediate early (IE) prot

105 As HCF1 is best known for its function in herpes simplex virus 1 (HSV-1) immediate early gene tran

106 teins have some functional similarities with herpes simplex virus 1 (HSV-1) immediate early protein I

107 tinocytes represent a primary entry site for herpes simplex virus 1 (HSV-1) in vivo.

108 Uncontrolled herpes simplex virus 1 (HSV-1) infection can advance to

109 Herpes simplex virus 1 (HSV-1) infection is an incurable

110 Herpes simplex virus 1 (HSV-1) infection is widespread a

111 f the intrinsic antiviral immune response to herpes simplex virus 1 (HSV-1) infection.

112 efects in IFN responses can result in lethal herpes simplex virus 1 (HSV-1) infections, usually from

113 Herpes simplex virus 1 (HSV-1) infects humans through st

114 Herpes simplex virus 1 (HSV-1) infects most people and c

115 The immediate early protein ICP0 of herpes simplex virus 1 (HSV-1) interacts with CIN85, an

116 We report here that UL37 of herpes simplex virus 1 (HSV-1) is a protein deamidase th

117 Human herpes simplex virus 1 (HSV-1) is a widespread pathogen,

118 Herpes simplex virus 1 (HSV-1) is among the most common

119 Infected cell protein 0 (ICP0) of herpes simplex virus 1 (HSV-1) is an alpha gene product

120 The UL16 tegument protein of herpes simplex virus 1 (HSV-1) is conserved among all he

121 Herpes simplex virus 1 (HSV-1) is one of the eight herpe

122 viral DNA from NPC-bound capsids.IMPORTANCE Herpes simplex virus 1 (HSV-1) is the causative agent of

123 We have shown previously that herpes simplex virus 1 (HSV-1) lacking expression of the

124 nal (NLS) sequences previously identified in herpes simplex virus 1 (HSV-1) large terminase and human

125 Herpes simplex virus 1 (HSV-1) latency entails the repre

126 s from a variant to target the mRNA encoding herpes simplex virus 1 (HSV-1) major transcription regul

127 Herpes simplex virus 1 (HSV-1) most commonly causes recr

128 ve previously shown that the live-attenuated herpes simplex virus 1 (HSV-1) mutant lacking the nuclea

129 During DNA encapsidation, herpes simplex virus 1 (HSV-1) procapsids are converted

130 Herpes simplex virus 1 (HSV-1) remodels nuclear membrane

131 ivating protein, p35, is important for acute herpes simplex virus 1 (HSV-1) replication in mice.

132 was to reexamine the requirement of UL21 for herpes simplex virus 1 (HSV-1) replication.

133 Infection of mature HD10.6 neurons by herpes simplex virus 1 (HSV-1) results in a delayed but

134 Ocular infection with herpes simplex virus 1 (HSV-1) sets off an inflammatory

135 sRNA, encephalomyocarditis virus (EMCV), and herpes simplex virus 1 (HSV-1) show impaired production

136 nd ICP34.5 are among the proteins encoded by herpes simplex virus 1 (HSV-1) that modulate type I IFN

137 Humans occasionally transmit herpes simplex virus 1 (HSV-1) to captive primates, who

138 e ability of the prototypic alphaherpesvirus herpes simplex virus 1 (HSV-1) to enter neurons via axon

139 Herpes simplex virus 1 (HSV-1) typically causes recurren

140 Herpes simplex virus 1 (HSV-1) UL20 plays a crucial role

141 The herpes simplex virus 1 (HSV-1) UL37 protein functions in

142 ogic protection requisite for an efficacious herpes simplex virus 1 (HSV-1) vaccine remain unclear wi

143 Herein, we report that the live-attenuated herpes simplex virus 1 (HSV-1) VC2 vaccine strain, which

144 Following infection of epithelial tissues, herpes simplex virus 1 (HSV-1) virions travel via axonal

145 Herpes simplex virus 1 (HSV-1), a leading cause of genit

146 lected TRIM proteins in autophagy induced by herpes simplex virus 1 (HSV-1), encephalomyocarditis vir

147 Persistent pathogens, such as herpes simplex virus 1 (HSV-1), have evolved a variety o

148 roinvasive alphaherpesviruses, such as human herpes simplex virus 1 (HSV-1), HSV-2, and veterinarian

149 trast, of the more than 80 mRNAs harbored by herpes simplex virus 1 (HSV-1), only 5 are spliced.

150 Following infection with herpes simplex virus 1 (HSV-1), PIAS1 is recruited to nu

151 Here we focus on two tegument proteins from herpes simplex virus 1 (HSV-1), pUL7 and pUL51, which ha

152 Although many herpesviruses, including herpes simplex virus 1 (HSV-1), stimulate mTORC1, how HS

153 zuelan equine encephalitis virus (VEEV), and herpes simplex virus 1 (HSV-1), suggesting that LIMK inh

154 as the replication of a related herpesvirus, herpes simplex virus 1 (HSV-1), was not impacted.

155 For herpes simplex virus 1 (HSV-1), we and others have previ

156 Herpes simplex virus 1 (HSV-1), which causes a variety o

157 Although most herpes simplex virus 1 (HSV-1)-infected individuals shed

158 esponse to restrict viral pathogens, such as herpes simplex virus 1 (HSV-1).

159 ganglia (TG) of mice latently infected with herpes simplex virus 1 (HSV-1).

160 SP141), bound to RIG-I during infection with herpes simplex virus 1 (HSV-1).

161 between AAV2 and one of its helper viruses, herpes simplex virus 1 (HSV-1).

162 f mice that have been ocularly infected with herpes simplex virus 1 (HSV-1).

163 choriomeningitis virus (LCMV) infection, or herpes simplex virus 1 (HSV1) infection was profoundly d

164 svirus [KSHV], Epstein-Barr virus [EBV], and herpes simplex virus 1 [HSV-1]) genomes and induces the

165 unoglobulin M/immunoglobulin G antibodies to herpes simplex virus 1 and 2, cytomegalovirus, Epstein-B

166 us agalactiae, cytomegalovirus, enterovirus, herpes simplex virus 1 and 2, human herpesvirus 6, human

167 ibition decreases replication of adenovirus, herpes simplex virus 1 and influenza A in cultured prima

168 ability to be infected by the herpesviruses herpes simplex virus 1 and murine herpesvirus 68 and the

169 Here, we have characterized the full-length herpes simplex virus 1 gB in a native membrane by displa

170 pted this method to rapidly deliver a 152 kb herpes simplex virus 1 genome cloned in yeast into mamma

171 nt productive infection of cultured cells by herpes simplex virus 1 has made it a paradigm for this m

172 e the molecular organization of chromatin in herpes simplex virus 1 infection and its effect on the t

173 155 expression was up-regulated after ocular herpes simplex virus 1 infection, with the increased Mir

174 promised survival during influenza virus and herpes simplex virus 1 infections.

175 er replication of BKV, whereas influenza and herpes simplex virus 1 replication were clearly reduced.

176 study focuses on two tegument proteins from herpes simplex virus 1 that are conserved in all herpesv

177 tein domains, as well as a truncated form of herpes simplex virus 1 thymidine kinase (HSV-TK).

178 wed evidence of acute herpesvirus infection; herpes simplex virus 1 was found most often.

179 tudy, the structural, material properties of herpes simplex virus 1 were investigated.

180 V] 6, 18%; HHV8, 6%; Epstein-Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%;

181 e, whereas infection with influenza A virus, herpes simplex virus 1, or cytomegalovirus induced a str

182 provision of antagomir-155 nanoparticles to herpes simplex virus 1-infected mice led to diminished S

183 Random walk modelling of herpes simplex virus 1-sized particles in a three-dimens

184 viously established that cells infected with herpes simplex virus 2 (HSV-2) are disrupted in their ab

185 Subunit vaccines based on the herpes simplex virus 2 (HSV-2) glycoprotein D (gD-2) hav

186 nital herpes prophylactic vaccine containing herpes simplex virus 2 (HSV-2) glycoproteins C (gC2) and

187 Several prophylactic vaccines targeting herpes simplex virus 2 (HSV-2) have failed in the clinic

188 Herpes simplex virus 2 (HSV-2) is a causative agent of g

189 Herpes simplex virus 2 (HSV-2) is a major global pathoge

190 ified virion host shutoff protein (vhs) as a herpes simplex virus 2 (HSV-2) protein capable of disrup

191 llions of people worldwide are infected with herpes simplex virus 2 (HSV-2), and to date, an efficaci

192 %; varicella zoster virus, 3%; HHV7, 2%; and herpes simplex virus 2, 1%.

193 ted with human immunodeficiency virus (HIV), herpes simplex virus 2, and CMV starting antiretroviral

194 Alpha herpesviruses, such as herpes simplex virus and pseudorabies virus (PRV), are n

195 5 on the structural stability of icosahedral herpes simplex virus capsids.

196 ital specimens each containing >/=105 copies herpes simplex virus DNA/ml collected a median of 5 mont

197 The herpes simplex virus for example evades immune surveilla

198 The transcription factor ICP4 from herpes simplex virus has a central role in regulating th

199 bitor ICP47, a small protein produced by the herpes simplex virus I.

200 h inhibits HIV-1, HIV-2, Influenza virus and herpes simplex virus infection, and enhances the potenti

201 ons, rubella, cytomegalovirus infection, and herpes simplex virus infections (TORCH), Apgar score <5

202 icase-primase inhibitor for the treatment of herpes simplex virus infections, was prepared.

203 r development of antiviral agents.IMPORTANCE Herpes simplex virus is a major pathogen, and although n

204 The capsid of herpes simplex virus is built up of a variety of protein

205 ens from entering the endoplasmic reticulum, herpes simplex virus is hidden from cytotoxic T lymphocy

206 ovir decreased the recurrence of any type of herpes simplex virus keratitis by approximately half.

207 nfections of the superficial cornea, such as herpes simplex virus keratitis or Acanthamoeba keratitis

208 us keratitis is significantly different from herpes simplex virus keratitis, and further studies usin

209 Further, in the murine model of herpes simplex virus keratitis, corneal pathology and ly

210 itis is significantly different from that of herpes simplex virus keratitis.

211 of autophagy/xenophagy results with mutated herpes simplex virus lacking its ICP34.5 neurovirulence

212 ) relocation of nonintegrin receptors (e.g., herpes simplex virus nectin1 and Kaposi's sarcoma-associ

213 h clinically diagnosed ARN, PCR-positive for herpes simplex virus or varicella zoster virus and evalu

214 iral reactivation, exhibiting parallels with herpes simplex virus reactivation.

215 of the lipid raft-dwelling US9 protein from Herpes Simplex Virus strikingly overlaps with that of th

216 transgenic mice expressing the suicide gene, herpes simplex virus thymidine kinase (HSVtk), driven by

217 g AAVP particles served to deliver the human Herpes simplex virus thymidine kinase type-1 (HSVtk) gen

218 Infectious titers of EBOV or herpes simplex virus type 1 (HSV-1) in detergents-treate

219 Lytic infection with herpes simplex virus type 1 (HSV-1) induces profound mod

220 mount of virus reactivation following ocular herpes simplex virus type 1 (HSV-1) infection.

221 Herpes simplex virus type 1 (HSV-1) is a leading cause o

222 Herpes simplex virus type 1 (HSV-1) is a ubiquitous viru

223 Glycoprotein D (gD) of herpes simplex virus type 1 (HSV-1) is one of four glyco

224 lly occurs during delivery from mothers with herpes simplex virus type 1 (HSV-1) or type 2 (HSV-2) ge

225 ological stress.IMPORTANCE Like all viruses, herpes simplex virus type 1 (HSV-1) reproduction relies

226 we reported a new series of highly defective herpes simplex virus type 1 (HSV-1) vectors that were fu

227 ormational approach to genome engineering of herpes simplex virus type 1 (HSV-1), which has a large D

228 Knockout of TRIM14 impairs herpes simplex virus type 1 (HSV-1)-triggered antiviral

229 Support for the concept that herpes simplex virus type 1 (HSV1), when present in the

230 Herpes simplex virus type 1 and Alzheimer's disease: pos

231 athway are associated with susceptibility to herpes simplex virus type 1 encephalitis (HSE).

232 We found wide distribution of O-glycans on herpes simplex virus type 1 glycoproteins and demonstrat

233 gene encoding the prodrug-converting enzyme herpes simplex virus type 1 thymidine kinase (HSV1-tk) i

234 CMV, herpes simplex virus type 1, and human herpesvirus 6 inf

235 e association of 4 human herpesviruses (CMV, herpes simplex virus type 1, human herpesvirus type 6, a

236 pathogens were detected by mNGS (4 cases of herpes simplex virus type 1, including 1 case of coinfec

237 Talimogene laherparepvec (T-VEC) is a herpes simplex virus type 1-derived oncolytic immunother

238 chnique for mapping O-glycosylation sites on herpes simplex virus type 1.

239 fumarate (TDF) has in vitro activity against herpes simplex virus type 2 (HSV-2) and reduced HSV-2 ac

240 We focused on herpes simplex virus type 2 (HSV-2) because prior publis

241 ndoms for protection against transmission of herpes simplex virus type 2 (HSV-2) has been examined in

242 Despite the high prevalence of herpes simplex virus type 2 (HSV-2) in sub-Saharan Afric

243 Genital herpes simplex virus type 2 (HSV-2) infection causes rec

244 Globally, herpes simplex virus type 2 (HSV-2) infection is the mos

245 mmune system's protective effect against one herpes simplex virus type 2 (HSV-2) infection protects a

246 or NK cell function in response to a mucosal herpes simplex virus type 2 (HSV-2) infection.

247 HIV and herpes simplex virus type 2 (HSV-2) infections cause a s

248 tor in clinical development for treatment of herpes simplex virus type 2 (HSV-2) infections.

249 Herpes simplex virus type 2 (HSV-2) reactivation is acco

250 ART) has been incompletely characterized for herpes simplex virus type 2 (HSV-2).

251 Herpes simplex virus type 2 (HSV-2; herpes) exacerbates

252 against cervical cancer, cervical dysplasia, herpes simplex virus type 2, chlamydia, and syphilis.

253 l among 3408 persons coinfected with HIV and herpes simplex virus type 2.

254 nd spinal cord in response to infection with herpes simplex virus type 2.

255 mydia trachomatis, Neisseria gonorrhoeae, or herpes simplex virus type 2.

256 lar functions affected by lytic infection of Herpes Simplex Virus type I in Human primary fibroblasts

257 f herpetic neuralgia using a murine model of Herpes Simplex Virus Type-1 (HSV-1) infection.

258 Herpes simplex virus type-1 (HSV-1) is one of the most w

259 CB) women, including bacterial vaginosis and herpes simplex virus type-2 (HSV-2) infection.

260 he impact of antiretroviral therapy (ART) on herpes simplex virus type-2 (HSV-2) replication is uncle

261 ded controls for sociodemographic variables, herpes simplex virus type-2 status, and recreational dru

262 rr virus (EBV), human herpesvirus 6 (HHV-6), herpes simplex virus types 1 (HSV-1) and 2 (HSV-2), and

263 Herpes simplex virus types 1 and 2 (HSV-1 and HSV-2) are

264 e = 11.1 weeks) to Epstein Barr virus (EBV), herpes simplex virus types 1 and 2 (HSV-1 and HSV-2), an

265 Trichomonas vaginalis and viral pathogens (herpes simplex virus types 1 and 2 and adenovirus) can c

266 Seroprevalence of EBV, CMV, herpes simplex virus types 1 and 2, and human herpesviru

267 ein Barr virus (EBV), cytomegalovirus (CMV), herpes simplex virus types 1 and 2, human herpesvirus 8)

268 iency virus, hepatitis B virus, and neonatal herpes simplex virus, from which lessons for the evaluat

269 ing of mucosal immunity and then compare the herpes simplex virus, human immunodeficiency virus, and

270 papilloma virus, hepatitis B and C viruses, herpes simplex virus, norovirus, rotavirus, parvovirus,

271 use substantial impairment: cytomegalovirus, herpes simplex virus, rubella virus, Toxoplasma gondii,

272 ded toxoplasmosis, rubella, cytomegalovirus, herpes simplex virus, syphilis, and human immunodeficien

273 e (CTL), Th1 cell, or Th17 cell responses to herpes simplex virus, Toxoplasma gondii, and Citrobacter

274 97%; PPV range, 52%-95%; NPV range, 79%-80%; herpes simplex virus, vulvar ulcerations: sensitivity, 2

275 Herpes simplex virus-1 (HSV-1) causes life-long morbidit

276 Herpes Simplex Virus-1 (HSV-1) is a ubiquitous human pat

277 Herein we demonstrate that oncolytic herpes simplex virus-1 (HSV-1) potently activates human

278 ed interferon-alpha secretion in response to herpes simplex virus-1 (HSV-1), whereas granzyme-B induc

279 76-9 (MHC I low) tumors respond to oncolytic herpes simplex virus-1 (oHSV-1) and PD-1 blockade combin

280 on library generated using the high-capacity herpes simplex virus-1 amplicon technology to deliver ba

281 ers in the lung, and are resistant to lethal herpes simplex virus-1 infection due to enhanced product

282 Here, we address this deficit, focusing on a herpes simplex virus-1 protein, ICP27.

283 sneuronal viral tract tracer, H129 strain of herpes simplex virus-1.

284 and generalized infections that can lead to herpes simplex virus-induced acute liver failure.

285 Furthermore, an unrelated herpes simplex virus-thymidine kinase (HSV-TK) promoter

286 r, and parvovirus B19), cytomegalovirus, and herpes simplex virus.

287 e fusion also proved to be well tolerated in herpes simplex virus.

288 ere independent of varicella-zoster virus or herpes-simplex virus 1 coinfection.

289 mmunoglobulin G seropositivity for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were

290 Herpes simplex viruses (HSV) are human pathogens that sw

291 significant effect.IMPORTANCE Infections by herpes simplex viruses (HSV) cause painful cold sores or

292 l B virus isolates to use entry receptors of herpes simplex viruses (HSV).

293 Herpes simplex viruses (HSVs) are human pathogens that c

294 Herpes simplex viruses (HSVs) are unusual in that unlike

295 A key property of herpes simplex viruses (HSVs) is their ability to establ

296 Human herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) are lar

297 Herpes simplex viruses 1 and 2 (HSV-1 and HSV-2) infect

298 rus, mumps virus, measles virus, lyssavirus, herpes simplex viruses 1 and 2, Epstein-Barr virus, ente

299 Modified herpes simplex viruses that are unable to produce glycop

300 domains from the Drosophila fushi tarazu and Herpes simplex VP16 was created.