ライフサイエンスコーパス: aphid

1 studied insects (e.g. lepidopteran larvae or aphids).

2 ed in virus uptake and transport through the aphid.

3 of Arabidopsis resistance to the green peach aphid.

4 und to display enhanced resistance to potato aphids.

5 along this continuum: aggressive mimicry in aphids.

6 aps throughout the year to collect migrating aphids.

7 paired when provided with pyrethroid-treated aphids.

8 nd in the phloem sap of fava beans fed on by aphids.

9 cae), indicating enhanced resistance against aphids.

10 mino acids, the major source of nitrogen for aphids.

11 role in basal resistance to insects, such as aphids.

12 ound that immune costs are limited to winged aphids.

13 th the specialized mode of phloem feeding by aphids.

14 n falling, cats rotate their body [8], while aphids [9] and ants [10, 11] manipulate wind resistance

15 or heritable symbiosis is the association of aphids, a clade of sap-feeding insects, and Buchnera aph

16 ther BIK1 functions in plant defense against aphids, a group of insects with a specialized phloem sap

17 mutually obligate symbiosis, between the pea aphid (Acyrthosiphon pisum) and its maternally transmitt

18 atings between two parental genotypes of pea aphid (Acyrthosiphon pisum) differing in virulence on a

19 We use the pea aphid (Acyrthosiphon pisum) to address this problem.

20 are also compromised in immunity to the pea aphid (Acyrthosiphon pisum), for which Arabidopsis is no

21 increase the fitness of their host, the pea aphid (Acyrthosiphon pisum), under natural conditions.

22 settling and survival on dodder vines by pea aphids (Acyrthosiphon pisum) were reduced significantly

23 The pea aphid, Acyrthosiphon pisum, maintains extreme variation

24 EMV) coat protein (CP) in the gut of the pea aphid, Acyrthosiphon pisum, using a far-Western blot met

25 et, namely as an effector protein in the pea aphid, Acyrthosiphon pisum.

26 y of fused proteins into the hemocoel of pea aphids, Acyrthosiphon pisum, without virion assembly.

27 in red and green colour morphs of clonal pea aphids, Acyrthosiphon pisum.

28 la, a gammaproteobacterium that colonized an aphid ancestor 150 million years ago and persists in alm

29 pe III secretion system, also reduced potato aphid and green peach aphid fecundity, respectively.

30 e show that controlling the genetics of both aphid and host can reveal novel recombinant genotypes wi

31 potential geographical distributions of the aphid and its steppe habitat.

32 tion of chemical insecticides to manage both aphids and aphid-vectored plant viral disease.

33 ssociated with population dynamics of cereal aphids and armyworms feeding on wheat in Henan province

34 cologically based pest management for cereal aphids and armyworms under global change.

35 yield per unit area) and damage from cereal aphids and armyworms were examined.

36 lants and analyzed the Bt protein residue in aphids and compared the effects of Bt plants and a pyret

37 ays an important role in plant resistance to aphids and heat stress.

38 by manipulating plant physiology to attract aphids and increase aphid reproduction.

39 Many plant viruses depend on aphids and other phloem-feeding insects for transmission

40 promising strategy for transgenic control of aphids and potentially other hemipteran pests.

41 s survived consumption of pyrethroid-treated aphids and that ovipositional behavior of A. colemani wa

42 rogen input and cropland expansion on cereal aphids and their natural enemies.

43 otton mealybug), Myzus persicae (green peach aphids) and Bemisia tabaci (silver leaf whitefly).

44 ension to the classical textbook paradigm of aphid-ant relationships by showcasing a complex system a

45 ediates the abundance and ant tending of the aphid Aphis helianthi feeding on the herb Ligusticum por

46 levels negatively impact virulence, and that aphids are able to reduce VPS52 levels during infestatio

47 iated with the population dynamics of cereal aphids are fertilizer input and mean temperature in Febr

48 In field populations, Hamiltonella-infected aphids are found at low to moderate frequencies and whil

49 Aphids are important herbivores of both wild and cultiva

50 We found that winged aphids are less resistant and mount a weaker immune resp

51 ables evolutionary and ecological expansion; aphids are one of many insect groups that would not exis

52 plants, vacuole localization disappears when aphids are removed, and this phenomenon occurs for anoth

53 Aphids are sap-feeding plant pests and harbor the endosy

54 Pea aphids are typically unwinged but produce winged offspri

55 Although aphids are worldwide crop pests, little is known about a

56 Inoculation of pea aphid Armet protein into tobacco leaves induced a transc

57 Pea aphid Armet's physical and chemical properties and its i

58 nfection, NIa-Pro expression alone increased aphid arrestment, suppressed callose deposition and incr

59 iphella stipae stipae is a xerothermophilous aphid, associated with Palaearctic temperate steppe zone

60 No aphid-associated molecular pattern has yet been identifi

61 ystemic release of volatiles by plants after aphid attack, reducing parasitic wasp recruitment and in

62 nera cells are densely packed in specialized aphid bacteriocyte cells.

63 By genome-wide association mapping of aphid behavior on 350 natural Arabidopsis thaliana acces

64 nscytosed across the gut epithelium into the aphid body cavity prior to release in saliva as the aphi

65 The aphid-Buchnera endosymbiosis provides a powerful system

66 Recent results for the aphid-Buchnera symbiosis and related systems illustrate

67 ong them serious pests (e.g. some species of aphid), but also many beneficial species (e.g. natural e

68 Solstice to aphids, but there was no effect on aphid development on

69 es that attract parasitic wasps, and the pea aphid can carry facultative endosymbiotic bacteria that

70 a plant RNA virus transmitted exclusively by aphids--causes disease in multiple food crops.

71 We measured fecundity of winged and unwinged aphids challenged with a heat-inactivated fungal pathoge

72 s that mir1-mediated resistance to corn leaf aphid (CLA; Rhopalosiphum maidis), a phloem sap-sucking

73 Aphid clones also vary in fitness, measured as developme

74 ch repeat protein that in addition to potato aphids confers resistance to two additional phloem-feedi

75 hese studies show considerable potential for aphid control, field trials employing the single and dou

76 Studies on aphids, crickets and planthoppers have revealed that alt

77 Cereal aphid damage has been rising, while armyworm damage had

78 receiving plants of comparable magnitude to aphid damage of neighbours, and the effects of the blend

79 communication to herbivore identity, as each aphid-damaged plant only induced resistance in neighbour

80 mount a weaker immune response than unwinged aphids, demonstrating that winged aphids pay higher cost

81 th of selection and indirectly by increasing aphid density and thus weakening genetic drift.

82 on low resistance plants, predators reduced aphid density by 35% and population growth by 86%.

83 henism, environmental stressors such as high aphid density cause asexual, viviparous adult female aph

84 ators had no impact on the number of nymphs, aphid density or population growth on high resistance pl

85 Aphid-derived elicitors induce expression of PHYTOALEXIN

86 lstice to aphids, but there was no effect on aphid development on this variety.

87 Host-associated races of pea aphid discriminate between plant species in race-specifi

88 most of the total effect of the predator on aphid dispersal and number of nymphs, the suppressive ef

89 ion risk rather than predation rate promoted aphid dispersal and varied with host plant resistance.

90 Aphid dispersal in response to predation risk was greate

91 Depending on the haplotype, aphids displayed a different duration of salivation in t

92 worldwide crop pests, little is known about aphid effector genes underlying virulence and avirulence

93 Domestication slowed aphid evolution by 13.5%, maintained 10.4% greater aphid

94 models suggested that domestication affects aphid evolution directly by reducing the strength of sel

95 sticity of genes that have duplicated during aphid evolution.

96 ifferential regulation of genes belonging to aphid-expanded gene families.

97 nduced resistance to insects and manipulated aphid exposure to lethal and risk predators.

98 peach aphid (Myzus persicae), and increases aphid fecundity compared with uninfected control plants.

99 Aphid fecundity was decreased significantly in TAO plant

100 m, also reduced potato aphid and green peach aphid fecundity, respectively.

101 trikingly, this effect was reversed when the aphids fed directly upon Arabidopsis, which indicates an

102 Aphids fed on these fusion proteins showed signs of neur

103 eral times higher in the salivary gland when aphids feed on bean plants than when they feed on an art

104 rmet transcript by RNA interference disturbs aphid feeding behavior on fava beans measured by the ele

105 were decreased significantly in response to aphid feeding in all the lines, the effect being the lea

106 tion of kaempferol 3,7-dirhamnoside, whereas aphid feeding induces resistance via a novel mechanism i

107 nd aphids is ongoing, the stealthy nature of aphid feeding makes both the mechanisms and outcomes of

108 Synthetic diet aphid feeding trial bioassays with recombinant Mir1-Cys

109 ody cavity prior to release in saliva as the aphid feeds.

110 Knock-down of cathepsin B genes reduced aphid fitness, but only on the host that induced upregul

111 ng parasitic wasp recruitment and increasing aphid fitness.

112 reads from the re-sequencing data of 33 pea aphid genomes from individuals specialized on different

113 We also identified strong aphid genotype x symbiont-strain interactions, such that

114 strategy against parasitoids varied for each aphid genotype; one performed best with no protective sy

115 We experimentally infected five aphid genotypes (two lowly and three highly resistant),

116 In susceptible aphid genotypes, parasitized sublines infected with Hami

117 evolution by 13.5%, maintained 10.4% greater aphid genotypic diversity and 5.6% higher genotypic rich

118 ae Sulzer, commonly known as the green peach aphid (GPA), which is an important phloem sap-consuming

119 Extract of the green peach aphid (GPA; Myzus persicae) triggers responses character

120 root symbionts that can increase or decrease aphid growth rates and reproduction, but the reason by w

121 sylated receptor aminopeptidase N in the pea aphid gut and is transcytosed across the gut epithelium

122 reviously demonstrated to bind to APN in the aphid gut and to impede PEMV uptake into the hemocoel; t

123 identification of a receptor protein in the aphid gut called aminopeptidase N, which is responsible

124 ment of proteins found on the surface of the aphid gut epithelium resulted in identification of this

125 ium resulted in identification of this first aphid gut receptor for a plant virus.

126 Finally, in assays with aphid gut-derived brush border membrane vesicles, bindin

127 ication of insecticides; however, over time, aphids have developed resistance to many insecticidal cl

128 psis (Arabidopsis thaliana), with or without aphid herbivory.

129 strains of endosymbiotic bacteria within an aphid host influence the outcome of symbiosis for both s

130 In some instances, aphid hosts suffered additional survival costs when host

131 e fungal infection, and we characterized the aphid immune response to fungi by measuring immune cell

132 AO activities were decreased in response to aphids in all genotypes.

133 Aphids, in addition to weakening plants by feeding on th

134 abidopsis alters ET responses and suppresses aphid-induced callose formation in an ET-dependent manne

135 e to wild-type plants, bik1 displayed higher aphid-induced hydrogen peroxide accumulation and more se

136 cost in terms of plant growth reduction than aphid-induced resistance.

137 hermore, a synthetic blend of the five major aphid-induced VOCs (ethanone, limonene, methyl salicylat

138 , Arabidopsis BIK1 confers susceptibility to aphid infestation through its suppression of PAD4 expres

139 ole of apoplastic redox state in controlling aphid infestation was explored using transgenic tobacco

140 identify AO-dependent mechanisms that limit aphid infestation.

141 To investigate the role of glycans in PEMV-aphid interactions and explore the possibility of viral

142 their reproduction, TuMV may influence plant-aphid interactions to promote its own transmission.

143 effects on the parasite itself and on dodder-aphid interactions.

144 The wing polyphenism of pea aphids is a compelling laboratory model with which to st

145 erm maintenance of symbiont co-infections in aphids is likely to be determined primarily by costs of

146 o-evolutionary adaptation between plants and aphids is ongoing, the stealthy nature of aphid feeding

147 toms, indicating bik1-mediated resistance to aphids is PAD4 dependent.

148 le of phloem proteins in plant resistance to aphids is still largely elusive.

149 la and Hamiltonella reduce the proportion of aphids killed by the specific natural enemies against wh

150 Changes in plant N concentration influenced aphid life history and behavior, and N concentration was

151 oat aphid (Rhopalosiphum padi), by examining aphid life history, feeding behavior and plant physiolog

152 Here we report the gall-forming aphid-like parasite phylloxera, Daktulosphaira vitifolia

153 in symbiont titer among genetically distinct aphid lines harboring the same Buchnera haplotype.

154 e placed experimental populations of two pea aphid lines, each with and without symbionts, in five we

155 ca oleracea, to different types of cues from aphid lion (Chrysoperla carnea).

156 rometry, we identified in saliva from potato aphids (Macrosiphum euphorbiae) 105 proteins, some of wh

157 red for Mi-1.2-mediated resistance to potato aphids (Macrosiphum euphorbiae).

158 population growth, density and dispersal of aphids (Macrosiphum euphorbiae).

159 We have recently found that individual aphids may employ each defence individually, occasionall

160 and indicate that the annual sexual cycle in aphids may lead to frequent novel genotypes with both in

161 The vetch aphid Megoura viciae feeds exclusively on the Fabaceae,

162 For the aphid Megoura viciae this effect was reversed under autu

163 , in their suppression of populations of the aphid Metopolophium dirhodum.

164 izer and cropland expansion benefited cereal aphids more than primary parasitoids and leaf-dwelling p

165 nd the strength of an immune response across aphid morphs that differ in life-history strategy but ar

166 henism is transgenerational, in that the pea aphid mother experiences the environmental signals, but

167 Certain races of the polyphagous aphid Myzus persicae have recently adapted to feed on to

168 ly important economic pest - the green peach aphid (Myzus persicae) - growing on 34 plant taxa, repre

169 Although susceptibility of aphid (Myzus persicae) infestation was similar in all li

170 feeding by its aphid vector, the green peach aphid (Myzus persicae), and increases aphid fecundity co

171 loperonospora arabidopsidis) and green peach aphid (Myzus persicae), but retained susceptibility to b

172 splayed reduced fecundity of the green peach aphid (Myzus persicae), indicating enhanced resistance a

173 we exposed four naive clones of green peach aphid (Myzus persicae), maintained on the model crop Bra

174 in Arabidopsis infested with the green peach aphid (Myzus persicae).

175 owever, settling and survival of green peach aphids (Myzus persicae) were not affected.

176 Remarkably, the green peach aphid, Myzus persicae, colonises plant species across 40

177 The peach potato aphid, Myzus persicae, is one of the most important agri

178 sitoid Aphidius colemani) of the green peach aphid, Myzus persicae.

179 on the Fabaceae, whereas the currant-lettuce aphid Nasonovia ribisnigri alternates hosts between the

180 otein-based effectors have been described in aphids, nematodes, fungi and bacteria.

181 However, pests such as aphids not susceptible to Cry proteins may require other

182 sk experiment also showed that the number of aphid nymphs increased in the presence of risk predators

183 Here, we report the first aphid OBP crystal structures and examine their molecular

184 nvestigated the additional survival costs to aphids of carrying multiple infections of symbiont speci

185 tionship with ants of the genus Tetramorium, aphids of the alternative morph are transported by the a

186 We fed aphids on Bt and non-Bt plants and analyzed the Bt prote

187 and double constructs showed no reduction in aphids or increase in parasitism.

188 how that two morphs clonally produced by the aphid Paracletus cimiciformis during its root-dwelling p

189 f ALAN on the population dynamics of a plant-aphid-parasitoid community with one plant species, three

190 n unwinged aphids, demonstrating that winged aphids pay higher costs for a less effective immune resp

191 gnificantly in the TAO plants in response to aphid perception relative to other lines.

192 tive and consumptive effects of predators on aphid performance and dispersal using a combination of p

193 showed Mendelian segregation consistent with aphid performance being controlled largely by a dominant

194 absence of BX13-dependent metabolites, while aphid performance increased, suggesting that DIM2BOA-Glc

195 o the direct effects of light environment on aphid performance, or indirectly through host plant qual

196 Here we show that the generalist aphid pest M. persicae is able to colonise diverse host

197 We show that when four aphid pest species-A. pisum, Rhopalosiphum padi, Aphis g

198 DD16 and NAO were shown to drive patterns in aphid phenology in a spatiotemporal context.

199 aim was to elucidate mechanisms that advance aphid phenology under climate change and explain these u

200 ey (RIS), elucidate the mechanisms advancing aphid phenology under climate change and show how by usi

201 ound that mycorrhizal colonisation increased aphid phloem feeding on T. monococcum MDR037 and MDR045,

202 (biting-chewing herbivore) and a specialist aphid (phloem feeder) differentially induce resistance a

203 pects of the molecular mechanisms underlying aphid-plant interactions are beginning to be understood.

204 However, a complete picture of aphid-plant interactions requires consideration of the e

205 that Armet is an effector protein mediating aphid-plant interactions.

206 sucking insects (order Hemiptera), including aphids, planthoppers, whiteflies and stink bugs, present

207 hid tending and, accordingly, ants increased aphid population growth in meadow but not understory env

208 mphs, the suppressive effect of predators on aphid population occurred largely through consumption.

209 We observed a reduction in aphid population size and increased feeding damage on no

210 ovided elevated levels of antibiosis (limits aphid population)- and antixenosis (deters aphid settlin

211 We expect potentially lower future aphid populations on noninfected plants but no change or

212 oninfected plants but no change or increased aphid populations on virus-infected plants therefore sub

213 ock plant virus transmission and to suppress aphid populations.

214 ion, potentially offers new tools to control aphid populations.

215 this hypothesis, Myzus persicae (green peach aphids) prefer to settle on Nicotiana benthamiana infect

216 On sli1 mutants, aphids prolonged their feeding sessions and ingested phl

217 , natural enemies were not affected when fed aphids reared on Bt broccoli, thus demonstrating the saf

218 anipulation, and plant receptors involved in aphid recognition.

219 ated with general acceptability of plants to aphids, regardless of host race.

220 Ant-aphid relationships represent an ideal system to this en

221 Plants fed upon by pea aphids release volatiles that attract parasitic wasps, a

222 tion of phloem-feeding insects, particularly aphids, remain poorly characterized.

223 Aphids represent a significant challenge to food product

224 is required for NIa-Pro's ability to enhance aphid reproduction on host plants, vacuole localization

225 t plant physiology and increased green peach aphid reproduction.

226 ssed in plants to determine their effects on aphid reproduction.

227 nt physiology to attract aphids and increase aphid reproduction.

228 (PAD4) into the bik1 background blocked both aphid resistance and HR-like symptoms, indicating bik1-m

229 troducing a loss-of-function mutation in the aphid resistance and senescence-promoting gene PHYTOALEX

230 to the HR-like symptoms or to the heightened aphid resistance associated with the bik1 mutant.

231 When competing for the same aphid resource, the feeding rate of A. bipunctata signif

232 t pest and virus vector, the bird cherry-oat aphid (Rhopalosiphum padi), by examining aphid life hist

233 The Russian wheat aphid (RWA), Diuraphis noxia Kurdjumov, is a major globa

234 The aphid RyR shares many of the features of other insect an

235 We show that potato aphid saliva and protein extracts induce the Mi-1.2 defe

236 Aphid saliva is predicted to contain proteins that modul

237 tinctively changes in the presence of potato aphid saliva, suggesting a model in which a constitutive

238 ecent advances include the identification of aphid salivary proteins involved in host plant manipulat

239 cated the potential for migration during the aphid season; the North Atlantic Oscillation (NAO) signa

240 During plant penetration and feeding, aphids secrete saliva that contains proteins predicted t

241 s aphid population)- and antixenosis (deters aphid settling)-mediated resistance to CLA compared with

242 se, loss of BIK1 function adversely impacted aphid settling, feeding, and reproduction.

243 New work in aphids shows that a nuclear-encoded protein resulting fr

244 of the first British annotated checklist of aphids since 1964.

245 the effect of AM fungi on the English Grain aphid (Sitobion avenae) development, reproduction, attra

246 iRNAs were identified from the English green aphid, Sitobion avenae (F.), of which 168 were conserved

247 is salicifolia and two ecologically distinct aphid species (one a dietary generalist and the other a

248 del species Drosophila melanogaster and four aphid species Acyrthosiphon pisum, Myzus persicae, Toxop

249 It is transmitted by numerous aphid species and causes a serious disease of cereal cro

250 an experimental community composed of three aphid species and their associated specialist parasitoid

251 toid community with one plant species, three aphid species and their specialist parasitoids.

252 light treatment reduced the abundance of two aphid species by 20% over five generations, most likely

253 etween these very similar OBP3s from the two aphid species is determined mainly by the direct pi-pi i

254 but not a non-protective) phenotype into one aphid species led to it being able to escape from its na

255 ften involves targeted responses to specific aphid species or even genotypes.

256 In addition, aphid species that attack multiple plants often carry di

257 We find that aphid species that have mutualistic associations with an

258 species occurs more frequently in unrelated aphid species that specialise on certain plant genera.

259 We use data from over a hundred aphid species to test if host life history is associated

260 is changed the relative density of the three aphid species which resulted in the extinction of the tw

261 different between plants attacked by the two aphid species, providing a putative chemical mechanism f

262 n years ago and persists in almost all 5,000 aphid species.

263 ed resistance in neighbours against the same aphid species.

264 While grafting and aphid stylectomy experiments have identified many macrom

265 logical variability that we see today in the aphid subspecies Ch. stipae stipae may in the future lea

266 show that, once in the brood chamber, mimic aphids suck on ant larva hemolymph.

267 rasitic wasp larva and thus markedly improve aphid survival after wasp attack.

268 han single infections, and actually improved aphid survival.

269 higher ant abundance and per capita rates of aphid tending and, accordingly, ants increased aphid pop

270 Aphids that are anholocyclic (permanently parthenogeneti

271 d in the performance of pyrethroid-resistant aphids that fed on Bt broccoli expressing Cry1Ab or Cry1

272 In aphids, the supply of essential amino acids depends on a

273 nsity cause asexual, viviparous adult female aphids to alter the developmental fate of their embryos

274 Yearly surveys consistently found aphids to be more than 17-fold more abundant on open mea

275 e discuss how host specialization can enable aphids to co-opt both the phytohormonal responses and de

276 These results not only add aphids to the limited list of arthropods known to biosyn

277 in diverse steps of viral infection, such as aphid transmission, polyprotein processing, and suppress

278 This is the first report of an aphid-transmitted geminivirus.

279 ome of approximately 13.4 kb and groups with aphid-transmitted viruses in the genus Cytorhabdovirus A

280 Aphids use chemical cues to locate hosts and find mates.

281 (Ebetaf), the alarm pheromone for many pest aphids, using a synthetic gene based on a sequence from

282 that NIa-Pro responds to the presence of the aphid vector during infection by relocalizing to the vac

283 To increase our understanding of plant virus-aphid vector interaction, we provide in vitro evidence s

284 efense induced in response to feeding by its aphid vector, the green peach aphid (Myzus persicae), an

285 virus pea enation mosaic virus into the pea aphid vector.

286 mical insecticides to manage both aphids and aphid-vectored plant viral disease.

287 we use the coat protein of a luteovirus, an aphid-vectored plant virus, to deliver a spider-derived,

288 nt of a nonchemical strategy for controlling aphid-vectored plant viruses to maximize food production

289 he chances for virus particle acquisition by aphid vectors and CaMV transmission.

290 By attracting aphid vectors and promoting their reproduction, TuMV may

291 ereal crops worldwide and are transmitted by aphid vectors.

292 However, the aphid-virus interactions required for disease transmissi

293 Uniquely, we detected Armet in aphid watery saliva and in the phloem sap of fava beans

294 No Bt protein residues in aphids were detected and no significant differences were

295 Early in the year when the first aphids were migrating, the effect of the winter NAO was

296 This indicated the aphids were not affected by the Cry proteins or the pyre

297 behavioural assays, three species of cereal aphids were repelled and foraging was increased for a pa

298 The aphids were then retrieved and mortality from parasitoid

299 e clusters collectively upregulate in single aphids within two days upon host switch.

300 tion of higher disease risk, and that winged aphids would be more resistant due to a stronger immune