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1 hogenic or likely pathogenic CNVs (0.08-18.9 Mb).
2  structure or redox properties of myoglobin (Mb).
3 ning a total of 55 848 composite parts (71.0 Mb).
4 udicots with a relatively small genome ( 260 Mb).
5  coincident punishment in the mushroom body (MB).
6 er than 2.5 Mb, and scaffolds longer than 15 Mb.
7 tioning affects odor-evoked responses in the MB.
8 he Gbetagamma-Akt-eNOS-sGC pathway to induce MB.
9 n, subjects received injections of saline or MB.
10 ssembly of its genome to a contig N50 of 2.5 MB.
11 ps on the GONR were removed by the reductive MB.
12 that result in the differential induction of MB.
13 of a new boronate-based fluorescent probe, 4-MB.
14 protein (APPL) is required for memory in the MB.
15 affold N50 of 8.7 Mb and the longest of 19.0 Mb.
16 to haplotype contigs with N50 greater than 7 Mb.
17 aban treated with PCCs for the management of MBEs.
18 rt blockers of the Carla Koehler laboratory (MB)-10 inhibited import of substrates that require the T
19 nd molecular dynamics modeling revealed that MB-10 binds to a specific pocket in the C-terminal domai
20                                        Thus, MB-10 is the first small molecule modulator to attenuate
21 o the membrane, but biochemical studies with MB-10 show that this region is required for binding to t
22 ed upon drug treatment in MDA-MB-231 and MDA-MB-157 cells (P < 0.01-0.001).
23                              TNBC cells (MDA-MB-157, MDA-MB-231, and Hs578T) were treated with bromod
24  with 392 traits and diseases, covering 73.9 Mb (2.2%) of the human genome.
25 , MCF-10A (breast epithelial cells), and MDA-MB 231 (breast cancer cells).
26 ced growth of MCF-7 (-36%, P < 0.05) and MDA-MB-231 (-66%, P < 0.01) tumors and, for the MCF-7 tumor,
27  decreased MCF-7 (hormone-sensitive) and MDA-MB-231 (hormone-insensitive) breast cancer cell viabilit
28 talk in both MDA-MB-468 (basal-like) and MDA-MB-231 (mesenchymal-like) TNBC cell lines in which NO in
29 at least two cancer cells, breast cancer MDA-MB-231 and human glioblastoma U87 cancer lines, was demo
30 icantly decreased upon drug treatment in MDA-MB-231 and MDA-MB-157 cells (P < 0.01-0.001).
31 pic mouse models of human breast cancer, MDA-MB-231 and T-47D, which morphologically correlate to mes
32 e-negative basal breast cancer cell line MDA-MB-231 and the luminal breast cancer cell line MCF7: a)
33 to the drug compared to 2D culture where MDA-MB-231 attained a drug-resistant tumor-initiating phenot
34 , we validated our findings in MCF-7 and MDA-MB-231 breast cancer cell lines, which harbor lower hsa-
35 proliferation and activated apoptosis in MDA-MB-231 breast cancer cells in vitro and in vivo.
36 n experimental tumor construct, MCF7 and MDA-MB-231 breast cancer cells were coinjected into the mamm
37 m the mammary fat pad after transfecting MDA-MB-231 breast cancer cells, while BMD cells were isolate
38  of 4T1.2 mammary tumor cells as well as MDA-MB-231 breast cancer cells.
39 on attenuates invasion and metastasis of MDA-MB-231 cells and prolongs life span of mice injected wit
40 ue study showed that increased growth of MDA-MB-231 cells by HDAC5 overexpression was reversed by con
41                                  C2-like MDA-MB-231 cells dominated in untreated animals, but C3-like
42           Tumors derived from MCNeuA and MDA-MB-231 cells had high LDLR expression and formed larger
43                Loss of PRMT5 or WDR77 in MDA-MB-231 cells leads to defects in alternative splicing, i
44       Finally, re-expression of c-JUN in MDA-MB-231 cells promoted radioresistance and abrogated miR-
45  TRAF2 activation in the more aggressive MDA-MB-231 cells was TNFalpha independent but involved the e
46  0.1 and 0.80 +/- 0.19 kPa for MCF-7 and MDA-MB-231 cells).
47 l carcinoma CNE2 cells and breast cancer MDA-MB-231 cells, where these tumor cells autocrinely produc
48 ude mice, which had been inoculated with MDA-MB-231 cells, with inhibitor 38u via oral administration
49 t alter the acetylation level of LSD1 in MDA-MB-231 cells.
50  xenograft model, miR-221-overexpressing MDA-MB-231 clones were more aggressive and resistant to mda-
51                                      The MDA-MB-231 embedded spheroid tumor model exhibited the most
52 introduce lattice-light sheet imaging of MDA-MB-231 human breast cancer cells genetically engineered
53 c nude mice bearing estrogen-independent MDA-MB-231 human breast cancer xenografts.
54                 ZDHHC3 ablation in human MDA-MB-231 mammary tumor cell xenografts reduced the sizes o
55 tively assess ATP, ADP, and pH levels in MDA-MB-231 metastatic cancer cells as a function of the loca
56 8delta in breast cancer cells, MCF-7 and MDA-MB-231 resulted in a reduced rate of cell proliferation.
57 l lines while the NP accumulated more in MDA-MB-231 than MCF-7 potentially due to binding of hyaluron
58 asion, and inhibited the tumor growth of MDA-MB-231 TNBC cell xenografts in the mammary fat pads of f
59 stigations revealed that the survival of MDA-MB-231 TNBC cells relied heavily on the BCL-2/BCL-XL sig
60 ically increased cisplatin inhibition of MDA-MB-231 triple-negative breast cancer cell proliferation
61 umor activity in the MV4;11 leukemia and MDA-MB-231 triple-negative breast cancer xenograft models in
62 his, in turn, impacted three-dimensional MDA-MB-231 tumor cell migration behavior.
63 d an additive reduction in the growth of MDA-MB-231 tumor xenografts.
64                 Mesenchymal and invasive MDA-MB-231 tumors display higher vascularization and less ma
65 erentiated extracellular matrix (ECM) in MDA-MB-231 tumors relative to T-47D tumors.
66 es using mice carrying orthotopic breast MDA-MB-231 tumors showed that the cyclic peptide preferentia
67 was significantly more effective against MDA-MB-231 tumors, whereas T-47D tumors showed no significan
68 mutant K46N increased lung metastases in MDA-MB-231 xenograft mouse models.
69 hich was similar to the negative control MDA-MB-231 xenografts (percent ID per gram, 0.42 +/- 0.051;
70  (18)F-FLT uptake were also observed for MDA-MB-231 xenografts (tumor-to-muscle ratio, 7.2 +/- 0.9 fo
71 lastoma), A549 (lung adenocarcinoma) and MDA-MB-231(breast cancer).
72 elevated LDL-C in human triple-negative (MDA-MB-231) and mouse Her2/Neu-overexpressing (MCNeuA) breas
73 in NOD/SCID mice harboring xenografts of MDA-MB-231, a triple-negative breast cancer line constitutiv
74                  TNBC cells (MDA-MB-157, MDA-MB-231, and Hs578T) were treated with bromodomain-contai
75 was evaluated in 3 breast cancer models: MDA-MB-231, MCF-7, and ZR-75-1.
76 mong them, triple-negative breast cancer MDA-MB-231, which has the highest xCT messenger RNA level, h
77 in 3D spheroids and extended survival of MDA-MB-231-bearing mice.
78  of hyaluronic acid to CD44 expressed by MDA-MB-231.
79  administered fluorescent tracers MB-402 and MB-301 to urinary ratios of sugar tracers lactulose and
80 f enterally administered fluorescent tracers MB-402 and MB-301 to urinary ratios of sugar tracers lac
81 found to have selective activity against MDA-MB-453 cells, a model of the luminal androgen receptor (
82 atment with HER-family inhibitors, while MDA-MB-453 was comparatively resistant.
83 feed-forward NOS2/COX2 crosstalk in both MDA-MB-468 (basal-like) and MDA-MB-231 (mesenchymal-like) TN
84 nced endogenous overexpression of TRPV4, MDA-MB-468 and HCC1569.
85 rease in endoplasmic reticulum stress of MDA-MB-468 cells with time and with increasing oxygen tensio
86 curred in a TNFalpha-dependent manner in MDA-MB-468 cells.
87 ent GFP gene silencing efficiency in GFP-MDA-MB-468 TNBC cells without any significant cytotoxicity.
88  MCF7/VP16, BT474, T47D, ZR-75-1, SKBR3, MDA-MB-468) than normal breast cells (MCF-10A) or cell lines
89 avities matching the size of methylene blue (MB(+)), a versatile organic molecule used for bio-recogn
90 ory studies, we report that methyl benzoate (MB), a naturally-occurring compound in many plants, may
91                              Methylene blue (MB), a traditional mitochondrial-targeting antioxidant,
92 ents involving up to 65 breakpoints and 60.6 Mb across four chromosomes, further defining rare catego
93     Increase of the excimer fluorescence for MBs after hybridization with DNA was up to 24-fold.
94 horothioate-modified loop domain (2Me/PSLOOP MBs), an architecture that elicits marginal levels of no
95 nd assembled the pomegranate genome with 328 Mb anchored into nine pseudo-chromosomes and annotated 2
96 on activities at different concentrations of MB and achieved a high correlation coefficient of R(2)=0
97  genome, coldspots have a median size of 2.1 Mb and are spatially clustered.
98 ing cells from a mouse model of SHH-subgroup MB and grew them as sphere cultures.
99 rs of magnitude larger than the prefactor of Mb and Hb ( approximately 10(9) s(-1)).
100 al features revealed analogous heme sites in MB and HB and the absence of low-spin (LS) to high-spin
101 ius prefactors associated with CO binding in Mb and Hb.
102                                     However, MB and HIFU are limited by the half-life of the contrast
103 cies between states change every few trials; MB and MF control predict different responses following
104 ility to contingency changes can distinguish MB and MF strategies, with human subjects utilizing a hy
105 d play a role in shifting subjects between a MB and MF strategy.
106 , corroborating the parallel contribution of MB and MF systems in reinforcement learning.
107                                              MB and SLC48A1 affect meat color.
108 line, USVL1VR-Ls, with a scaffold N50 of 8.7 Mb and the longest of 19.0 Mb.
109 interaction interface between homodimeric CC MBS and homodimeric LZ PKG-Ialpha.
110 g the separation function, the binding of HP-MBs and SG could be avoided while a low background was a
111 oxy), and O2-bound (oxy) hemes in myoglobin (MB) and hemoglobin (HB) solutions and in porphyrin compo
112 ing in other heme systems such as myoglobin (Mb) and hemoglobin (Hb).
113 enced the genomes of T. cruzi Y strain (35.5 Mb) and three benznidazole-resistant clones derived from
114 chenko-Scheffler (TS), many-body dispersion (MBD), and random-phase approximation (RPA) approaches).
115 er than 100 kb, phase blocks longer than 2.5 Mb, and scaffolds longer than 15 Mb.
116 ral activities of early labile memory in the MB are consolidated into stable LTM at a few postsynapti
117                                          The MBs are composed of 2'-O-methyl RNAs with a fully phosph
118 the exception of those of the mushroom body (MB), are decommissioned by the ecdysone receptor and med
119                         We review the use of MBS as a treatment for obesity and obesity-related condi
120 herefore utilized the echoes from individual MBs as microscopic sensors of slow flow associated with
121 he detection of AZAs, using protein G-coated MBs as supports for antibody immobilisation and peroxida
122                        Using methylene blue (MB) as an electrochemical probe and differential pulse v
123 ) and organic pollutants (using methyl blue (MB) as an example) removal, and the removal mechanism wa
124 of Dengue virus (DENV) using methylene blue (MB) as an intercalating agent.
125 administration of PCCs for the management of MBEs associated with rivaroxaban or apixaban is effectiv
126 sducing surface and the dual-function biotin-MB-AuNPs bio-label, provides a simple and robust approac
127  integrated, dual function bio-label (biotin-MB-AuNPs) for both biorecognition and signal generation.
128                         The activated biotin-MB-AuNPs/miRNA complexes become available for capture, v
129 mly selected half-mouth quadrants (six sites/MB-B-DB/ MB-B-DL/MB-DB-ML-DL); and 4) the community peri
130 ed using a 15% cut-off point: 1) full-mouth (MB-B-DB/MB-B-DL); 2) two diagonal quadrants (six sites/M
131 -B-DL); 2) two diagonal quadrants (six sites/MB-B-DB/MB-B-DL); 3) two randomly selected half-mouth qu
132                         PMPEs that evaluated MB-B-DL sites of randomly selected half-mouth quadrants
133  a 15% cut-off point: 1) full-mouth (MB-B-DB/MB-B-DL); 2) two diagonal quadrants (six sites/MB-B-DB/M
134 2) two diagonal quadrants (six sites/MB-B-DB/MB-B-DL); 3) two randomly selected half-mouth quadrants
135 ted half-mouth quadrants (six sites/MB-B-DB/ MB-B-DL/MB-DB-ML-DL); and 4) the community periodontal i
136                             A magnetic bead (MB)-based direct immunoassay for the detection of AZAs,
137                     We then report the first MB-based imaging of intracellular dynamics and localizat
138                                          The MB-based immunoassay facilitated the quantification of a
139  envision the proposed minimally-engineered, MB-based technology for live-cell single-molecule RNA im
140 g a hybrid strategy that shifts towards more MB behavior over blocks, consequently corresponding to a
141 ur but rather displayed a shift towards more MB behavior over the first two blocks that was not attri
142 eins containing a methyl-CpG-binding domain (MBD) bind methylated DNA and interpret epigenetic marks,
143            The potential mechanisms by which Mb binding of alpha,beta-unsaturated aldehydes affect Mb
144 formoterol induces mitochondrial biogenesis (MB), but other beta2AR agonists, such as clenbuterol, do
145 PKA agonist pair N6/8-AHA (8-AHA-cAMP and N6-MB-cAMP) and treatment with endogenous activators of PKA
146                    Further, we showed that 4-MB can be potentially employed to visualize exogenous an
147                               We showed that MBs can detect single transcripts containing as few as 8
148  focused ultrasound (HIFU) and microbubbles (MBs) can improve tumor drug delivery from non-thermosens
149 n of ultrasound contrast agent microbubbles (MB) causes localized blood-brain barrier (BBB) disruptio
150 therapy, our structural understanding of the MBS CC interaction with LZ PKG-1alpha has remained limit
151 mal cerebellar granule (precursor) cells and MB cells not derived from granule precursors, only small
152 re positioning because many of its large ( 2 Mb) centromeres are not dominated by satellite DNA.
153 o the drain current difference caused by the MB concentration change).
154 hylation and methyl-cytosine binding domain (MBD) containing proteins are found throughout all verteb
155             These results suggest Drosophila MBD-containing proteins are required within the OA neura
156               Here, we report the Drosophila MBD-containing proteins, dMBD-R2 and dMBD2/3, contribute
157 both MB substrate and maintenance methylated MB corresponds to de novo methylation event.
158                           Prior to this work MB could only adapt from one generation to the other, so
159 neral bone disorder associated with CKD (CKD-MBD) could help the medical community to better understa
160 he probe which results in an increase in the MB current.
161 der (MBES) data from an AUV, forward-looking MBES data from ROVs and ROV-acquired videos to examine w
162 ined sideways-looking multibeam echosounder (MBES) data from an AUV, forward-looking MBES data from R
163 -mouth quadrants (six sites/MB-B-DB/ MB-B-DL/MB-DB-ML-DL); and 4) the community periodontal index.
164 kage analysis identified an approximately 18 Mb disease-associated locus on chromosome 4 (maximal log
165 o-buccal [DB], mid-buccal [B], mesio-buccal [MB], disto-lingual [DL], mid-lingual, and mesio-lingual
166 cluding a large topologically associated 1.2-Mb domain conserved in humans and mice that encompasses
167 r that restricts the dynamic mobility of the MBD domain.
168 panded the C. elegans reference genome by >2 Mb due to a more accurate determination of repetitive se
169  constructed directed assemblies of the 4.6 Mb E. coli genome, 48.5 kb lambda genome, and a represe
170 re determined for a range of thresholds, and MBS (ED(2) x DF) were calculated.
171  Assessed by in vitro flow chamber, targeted MB exhibited increased adhesion to platelets as compared
172                    The studies showed that 4-MB exhibits a dual ratiometric and calorimetric response
173 hanced lipid oxidation compared to wild-type Mb exposed to HNE.
174 er members of the methyl-CpG-binding domain (MBD) family, MeCP2 functions through the recognition of
175 highly concordant with large-scale (order 10 Mb) features of previously reported genetic maps for mou
176 ave thoroughly demonstrated the utility of 4-MB for intracellular peroxynitrite imaging.
177 trometry (PCR/ESI-MS) and molecular beacons (MB), for detecting genes conferring resistance/susceptib
178                                      The 353 Mb genome contains 35 597 predicted protein-coding genes
179 n this study, we report a high-quality 313.4-Mb genome sequence of a bottle gourd inbred line, USVL1V
180             Its rapid life cycle, small (440-Mb) genome, and advanced germplasm resources make birch
181 plex gene family, the DNA sequence of a 1.75-Mb genomic region spanning the Gli-2 locus was analyzed
182 hed both at short (<500 kb) and longer (>1.5 Mb) genomic distances.
183 ns separated by large genomic distances (>50 Mb) have yet to be characterized.
184                           Molecular beacons (MBs) have recently emerged as efficient probes for inter
185                               A 3:1 ratio of Mb:Hb promoted lipid oxidation similarly compared to add
186  similarly compared to adding a 1:1 ratio of Mb:Hb to washed muscle.
187                        In our design, the HP-MBs here serve together as the T4 PNK, DNA polymerase, a
188 ence of the MeCP2 methyl-CpG-binding domain (MBD), however, DNA methylation of decoys substantially (
189 siologic circuit that controls quiescence of MB-HSCs and hematopoietic progenitors marked by histidin
190 d cells lie in close anatomical proximity to MB-HSCs and produce histamine, which activates the H2 re
191 haride (LPS) treatment specifically recruits MB-HSCs and progenitors into the cell cycle; cycling MB-
192 and progenitors into the cell cycle; cycling MB-HSCs fail to revert into quiescence in the absence of
193 heir depletion, while an H2 agonist protects MB-HSCs from depletion after sepsis.
194 hysiological demands to intrinsically primed MB-HSCs to enforce homeostasis.
195 istamine, which activates the H2 receptor on MB-HSCs to promote their quiescence and self-renewal.
196     Myeloid-biased hematopoietic stem cells (MB-HSCs) play critical roles in recovery from injury, bu
197 this 3D skin model further demonstrated that MB improved skin viability, promoted wound healing and i
198     We identified 32 rare CNVs larger than 1 Mb in 31 patients.
199 y of large clonal autosomal mosaic events >2 Mb in size in the aging population.
200 guidelines have been proposed for the use of MBS in persons with type 2 diabetes.
201  a near-Nernstian potentiometric response to MB(+) in the nano- to micro-molar range.
202                Electroanalysis of myoglobin (Mb) in 10 plasma samples of healthy donors (HDs) and 14
203 bined to calculate mechanobehavioral scores (MBS) in women with (+) and without (-) bilateral TMJ dis
204 d source) to deliver agents (methylene blue, MB, in PBS) into bovine AC.
205 lity of the PMMA-NPs to promote the survivin-MB internalization, suggesting that this complex might r
206  one of the two genes in the approximately 1 Mb interval and the only gene disruption shared between
207 uencing to fine map it to an approximately 1 Mb interval.
208                                              MB is a unique material; one of its crucial applications
209                                         pFUS+MB is being considered as part of a noninvasive adjuvant
210                                      Because MB is considered an environment-friendly, it has great p
211                                              MBS is superior to medical therapy in reducing hyperglyc
212            In Drosophila, the mushroom body (MB) is critically involved in olfactory classical condit
213            In Drosophila, the mushroom body (MB) is the major site of associative learning.
214                             Medulloblastoma (MB) is the most common paediatric brain tumour that aris
215 h a robust immunosandwich format employing a MB-labelled detection antibody as a non-enzymatic report
216                                          The MB-LATER score (Male, Bundle brunch block, Left atrium >
217                            Validation of the MB-LATER score in other cohorts is underway.
218  reciprocal duplication, and the smaller 1.5 Mb LCR22A-B 22q11.2 deletion carry inversions of LCR22B-
219 rate that parents transmitting the de novo 3 Mb LCR22A-D 22q11.2 deletion, the reciprocal duplication
220             Metabolic and bariatric surgery (MBS) leads to weight loss in obese individuals and reduc
221 ormation to the central brain mushroom body (MB) learning/memory center.
222 rom which an algorithm-guided 192-member 4.5 Mb library is built.
223 et of hydrophobic and charged residues of CC MBS (localized within and adjacent to the C-terminal reg
224  Of these DMCs, 52% were located in one 15.5-Mb locus on chromosome 13, which encompassed the Bhmt ge
225 tanding of the binding restraints of this CC MBS.LZ PKG-Ialpha low-affinity heterotetrameric complex
226 ated high levels of spatial heterogeneity in MB, malignant glioma, and renal cell carcinoma (RCC).
227 NE and HHE (4-hydroxy-2-hexenal) facilitated Mb-mediated lipid oxidation similarly.
228 her, subjects did not remain at one level of MB/MF behaviour but rather displayed a shift towards mor
229  found that human subjects employed a hybrid MB/MF strategy on the task, corroborating the parallel c
230 ricated with a thiolated and methylene blue (MB)-modified oligo-adenine (A)-guanine (G) DNA probe.
231                                              MB NBs continue to express Imp into pupation, and the pr
232 nduction of transgenic Imp prevents many non-MB NBs from decommissioning in early pupae.
233 ocytosis but impairs sustained exocytosis in MB neurons and alters specific motor functions of 1-year
234  impairment in SV trafficking selectively in MB neurons but not cortical neurons caused by two PARK g
235 ly or optogenetically produced plasticity in MB neurons, altering their responses to odorants.
236 obust cAMP-dependent plasticity in intrinsic MB neurons, which is biased toward naturalistic reward l
237 nd sufficient for normal memory in intrinsic MB neurons.
238 abled the anchoring of 100 Mb of WGS and 420 Mb of BAC sequences, an exploration of genetic diversity
239 DB1-dependent loop formations bypassed 0.2-1 Mb of linear genome and radiated from the TAD(cPcdh) fri
240              Among them, approximately 71.43 Mb of novel CNVRs were detected in the Chinese cattle po
241 scanned 93 domestication sweeps occupying 74 Mb of the A subgenome and 104 Mb of the D subgenome, and
242 s occupying 74 Mb of the A subgenome and 104 Mb of the D subgenome, and identified 19 candidate loci
243 and increase in BMI by 0.14 kg/m(2) for each Mb of total deletion burden (P = 2.5 x 10(-10), 6.0 x 10
244 his genetic map enabled the anchoring of 100 Mb of WGS and 420 Mb of BAC sequences, an exploration of
245 Despite possessing the smallest genome (0.27 Mb) of any organism not subsisting within a host cell, t
246 for the management of major bleeding events (MBEs) on rivaroxaban or apixaban.
247 gies produced a draft nuclear genome of 36.1 Mb, organized into 321 scaffolds with L50 of 31 and N50
248 n, each make multiple en passant synapses to MB output neurons (MBONs) in each compartment.
249 g of alpha,beta-unsaturated aldehydes affect Mb oxidation and the onset of lipid oxidation are discus
250 y indices (BI; average:4,9), 94%(n = 161) of MB patients were identified by UCP-LFA and 78%(n = 133)
251  In the Bangladeshi cohort, including mainly MB patients with low BI (average:1), 41%(n = 14) and 44%
252 NIC HEDGEHOG (SHH)-subgroup medulloblastoma (MB) patients.
253 eful biomarker for detecting multibacillary (MB) patients.
254              We used it to interrogate the 2-Mb POU5F1 locus in human embryonic stem cells, and ident
255 cific biotinylated DNA/LNA molecular beacon (MB) probe was conjugated with gold nanoparticles (AuNPs)
256   Invertebrates generally have only a single MBD protein, MBD2/3, that does not always contain approp
257 lts suggest that when decoys are methylated, MBD proteins can block them and thereby allow Egr-1 to a
258           Square wave voltammetry signals of MB provided quantitative measurements of HBsAg with a li
259 , 89%) and 92% (85%, 97%) for PCR/ESI-MS and MB, respectively.
260 , 98%) and 88% (80%, 94%) for PCR/ESI-MS and MB, respectively.
261 MP signaling may serve to modulate intrinsic MB responses toward salient stimuli.
262 64)Cu-NODAGA-PSMA-IgG and (64)Cu-NODAGA-PSMA-Mb retained the ability to bind cell surface PSMA, and b
263 nfusion of fluorescent beads 3 d before pFUS+MB revealed the infiltration of CD68(+) macrophages at 6
264                               alphaLA-RL and Mb-RL form surfactant-saturated complexes above 5.6 and
265                                          For Mb-RL, the analysis gives complexes of two connected mic
266 its, or compartments, that compose the adult MB's alpha lobe, using a dataset of isotropic 8 nm voxel
267                                We identify a Mb-scale genomic region present in weedy rice but not cu
268  oxide nanoribbon (GONR) for methylene blue (MB) sensing.
269 rface of MCF-7 cells, the aptamer conjugated MBs showed a predominant capability for cell capture wit
270 in a concentration-dependent decrease in the MB signal.
271                    We sought to identify the MB signaling pathway of formoterol and the differences i
272 proliferative fitness ( approximately 3% per Mb), slowed tumor growth, and reduced metastatic progres
273 l target for therapy and a Molecular Beacon (MB) specific for survivin mRNA is available.
274    Like many draft genomes, however, the 158-Mb Spirodela genome sequence has not been resolved to ch
275 ower, prospective goal-directed model-based (MB) strategy and a fast, retrospective habitual model-fr
276 l difference between SacII digestion of both MB substrate and maintenance methylated MB corresponds t
277                           Results using H97A Mb suggested that the combination of HNE and low hemin a
278 his method to efficiently deliver large (1.1 Mb) synthetic yeast centromeric plasmids (YCps) to cultu
279 idation more readily than oxidative forms of Mb that retain their protoporphyrin moiety.
280 ome assemblies, of which sizes vary from 120 Mb to 3.5 Gb.
281 positive charge is proposed to transfer from MB to GONR during sensing.
282                               Application of MB to this 3D skin model further demonstrated that MB im
283 re, we report results from the phase 2 COMBI-MB trial.
284 eas pharmacological memory enhancement using MB was an effective strategy, but only for individuals w
285       Based on our laboratory toxicity data, MB was at least 5 to 20 times more toxic than the conven
286                                Additionally, MB was effectively applied to normal and cancer cell lin
287 ndrial-targeting antioxidants, we found that MB was more effective in stimulating skin fibroblast pro
288 structed 3D human skin model, indicated that MB was safe for long-term use, and did not cause irritat
289  presence of T4 PNK, the 3'-phosphoryl of HP-MBs was hydrolyzed to 3'-hydroxyl, thus serving as prime
290 sorption column, to pretreat methylene blue (MB) wastewater with high concentration ( 100 mg L(-1) or
291             Significantly larger ED, DF, and MBS were shown in women with compared to women without b
292                                      Daytime MBS were significantly larger (all P < 0.04) by up to 8.
293 particles coated magnetic microbeads (Au NPs-MBs), which were prepared through a novel and simple met
294 ic distance range ( approximately 700 kb-1.5 Mb), while interactions involving actively marked DNase
295 itioning modulate olfactory responses in the MB with in vivo imaging.
296 sequent combination of the immunorecognition MBs with 8-electrode arrays enabled the multiplexed elec
297  the rapid and efficient collection given by MBs with the good affinities to attach probe molecules e
298 oximately 1.2-Gb genome (scaffold N50 = 1.88 Mb) with at least 26 723 predicted genes for E. ulmoides
299 n of anionic mediator, i.e., methylene blue (MB) with free guanine (3'G) of ssDNA.
300 osphatase component, myosin-binding subunit (MBS), with the N-terminal LZ domain of protein kinase G

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