ライフサイエンスコーパス: LBBB

1 5% of subjects had left bundle-branch block (LBBB).

2 and patients with left bundle branch block (LBBB).

3 ight (RBBB) versus left bundle branch block (LBBB).

4 her complicated by left bundle-branch block (LBBB).

5 (MI) patients with left bundle-branch block (LBBB).

6 c HF patients with left bundle branch block (LBBB).

7 tients with LBBB or matched patients without LBBB.

8 rvedilol was seen in patients with CRT-D and LBBB.

9 %) developed RBBB, but no patients developed LBBB.

10 influence the mortality risk of TAVI-induced LBBB.

11 dle branch block (LBBB) versus those without LBBB.

12 een patients who did and did not develop new LBBB.

13 total of 233 patients (34.3%) developed new LBBB.

14 at AV delays of 40 ms was no different from LBBB.

15 7) were independent predictors of persistent LBBB.

16 th p < 0.001) compared with patients without LBBB.

17 MSI for detecting CAD in DCM with or without LBBB.

18 andard WMSI, particularly in the presence of LBBB.

19 sexes with LBBB, but not in patients without LBBB.

20 sponse to CRT-D therapy in patients with non-LBBB.

21 oximal LAD occlusions should cause RBBB, not LBBB.

22 roximal LAD septal perforator caused RBBB or LBBB.

23 better than QRS duration or the presence of LBBB.

24 t bundle-branch block (LBBB) and 308 had non-LBBB.

25 d more scar (2 [2-5] segments) compared with LBBB-1 and LBBB-2 (both 0 [0-1], P<0.05).

26 chronization therapy were most pronounced in LBBB-1 and worst in LBBB-3 patients.

27 ft ventricular free wall resulted in pattern LBBB-1.

28 septal deformation patterns were identified: LBBB-1=double-peaked systolic shortening (n=28); LBBB-2=

29 In 24 canine hearts with LBBB (12 acute, 6 with heart failure, and 6 with myocard

30 (2 [2-5] segments) compared with LBBB-1 and LBBB-2 (both 0 [0-1], P<0.05).

31 This transformed into pattern LBBB-2 by additionally simulating septal hypocontractili

32 -1=double-peaked systolic shortening (n=28); LBBB-2=early systolic shortening followed by prominent s

33 population comprised 533 CRT-D patients with LBBB, 212 (40%) with complete left-sided reverse remodel

34 re 1281 (70%) with left bundle-branch block (LBBB), 228 (13%) with right bundle-branch block, and 308

35 g septal hypocontractility, and into pattern LBBB-3 by imposing additional left ventricular free wall

36 were most pronounced in LBBB-1 and worst in LBBB-3 patients.

37 LBBB-3 revealed more scar (2 [2-5] segments) compared wi

38 by prominent systolic stretching (n=34); and LBBB-3=pseudonormal shortening with less pronounced late

39 B, 5+/-2 versus 1+/-1; P=0.0004; NICD versus LBBB, 4+/-2 versus 1+/-1; P=0.001); (2) evidence of earl

40 ior or anterior fascicles: narrow QRS versus LBBB, 5+/-2 versus 1+/-1; P=0.0004; NICD versus LBBB, 4+

41 ents were performed in 22 dogs, 9 with acute LBBB, 7 with chronic LBBB combined with infarction (embo

42 nt, T or Q wave or left bundle branch block (LBBB) abnormalities between the prehospital and initial

43 LV pacing with short AV delay and intrinsic LBBB activation accurately predicted the optimal AV dela

44 traction pattern assessment to identify true LBBB activation provided important prognostic informatio

45 rain echocardiography (2DSE) may detect true LBBB activation.

46 registry of high-volume centers, persistent LBBB after CoreValve Revalving System transcatheter aort

47 transient or absent) LBBB after TAVI with a balloon-expandable valve and its

48 pact of persistent left bundle-branch block (LBBB) after surgical aortic valve replacement.

49 ts with LLk and 72 consecutive patients with LBBB, all without prior myocardial infarction or sternot

50 th a lower mortality risk in both sexes with LBBB, although more pronounced among women.

51 mortality was 37.8% (n=88) in patients with LBBB and 24.0% (n=107) in patients without LBBB (P=0.002

52 were included in the study; 216 (43.5%) had LBBB and a QRSd >/=150 ms, 85 (17.1%) had LBBB and QRSd

53 ad LBBB and QRSd <150 ms, 92 (18.5%) had non-LBBB and a QRSd >/=150 ms, and 103 (20.8%) had non-LBBB

54 ith an improvement in survival in those with LBBB and a QRSD >/=180 ms (adjusted HR for death: 0.78;

55 95% CI: 0.68 to 0.91), but not in those with LBBB and a QRSD 150 to 179 ms (adjusted HR for death: 1.

56 in a community-based cohort of patients with LBBB and acute cardiopulmonary symptoms.

57 Both LBBB and CAD may do so by prolonging the total isovolumi

58 elf determined by the presence or absence of LBBB and CAD.

59 nalysis showed that among 1000 patients with LBBB and chest pain, 929 would survive without major str

60 CRT-D patients with LBBB and complete left-sided reverse remodeling had a si

61 io, 3.79; confidence interval, 2.95-4.87 for LBBB and hazard ratio, 3.53; confidence interval, 2.14-5

62 d concentric remodeling), and 6 with chronic LBBB and heart failure (rapid pacing, LBBB+HF, and eccen

63 In LBBB and LBBB+HF animals, endocardial conduction was app

64 LBBB and NICD patients had similar right ventricular tot

65 on fraction-matched control subjects without LBBB and no CAD (group B), and 10 normal control subject

66 Patients with persistent LBBB and no PPI at hospital discharge had a higher incid

67 ar synchrony) was observed for patients with LBBB and normal QRSd.

68 or the evaluation of the impact of new-onset LBBB and periprocedural PPI post-TAVR were sourced, resp

69 ated with better survival in both sexes with LBBB and QRS >/=130 ms, whereas there was no clear relat

70 In patients with non-LBBB and QRS >/=160 ms, the hazard ratio for the primary

71 duration of 150 ms or greater, compared with LBBB and QRS duration less than 150 ms or no LBBB regard

72 of 150 ms or greater (20.9%), compared with LBBB and QRS duration of 120 to 149 ms (26.5%; adjusted

73 30.7%; HR, 1.34 [99% CI, 1.20-1.49]), and no LBBB and QRS duration of 120 to 149 ms (32.3%; HR, 1.52

74 of 150 ms or greater (38.6%), compared with LBBB and QRS duration of 120 to 149 ms (44.8%; adjusted

75 45.7%; HR, 1.16 [99% CI, 1.08-1.26]), and no LBBB and QRS duration of 120 to 149 ms (49.6%; HR, 1.31

76 ar mortality were lowest among patients with LBBB and QRS duration of 150 ms or greater (20.9%), comp

77 rd ratio [HR], 1.30 [99% CI, 1.18-1.42]), no LBBB and QRS duration of 150 ms or greater (30.7%; HR, 1

78 mission were also lowest among patients with LBBB and QRS duration of 150 ms or greater (38.6%), comp

79 ; adjusted HR, 1.18 [99% CI, 1.10-1.26]), no LBBB and QRS duration of 150 ms or greater (45.7%; HR, 1

80 ing CRT-D implantation in clinical practice, LBBB and QRS duration of 150 ms or greater, compared wit

81 then categorized as having either LBBB or no LBBB and QRS duration of either 150 ms or greater or 120

82 of 150 ms or longer compared with those with LBBB and QRS of 120 to 129 ms was similar between sexes

83 Compared with women with LBBB and QRS of 120 to 129 ms, women with LBBB and QRS o

84 th LBBB and QRS of 120 to 129 ms, women with LBBB and QRS of 140 to 149 ms had a 27% lower mortality

85 Mortality in LBBB and QRS of 150 ms or longer compared with those wit

86 REVERSE demonstrated that LBBB and QRS prolongation are markers of reverse remodel

87 ection fraction) was better in patients with LBBB and QRSd >/=150 ms (12 +/- 12%) than in those with

88 with LBBB and QRSd <150 ms (8 +/- 10%), non-LBBB and QRSd >/=150 ms (5 +/- 9%), and non-LBBB and QRS

89 -LBBB and QRSd >/=150 ms (5 +/- 9%), and non-LBBB and QRSd <150 ms (3 +/- 11%) (p < 0.0001).

90 Sd >/=150 ms (12 +/- 12%) than in those with LBBB and QRSd <150 ms (8 +/- 10%), non-LBBB and QRSd >/=

91 ad LBBB and a QRSd >/=150 ms, 85 (17.1%) had LBBB and QRSd <150 ms, 92 (18.5%) had non-LBBB and a QRS

92 nd a QRSd >/=150 ms, and 103 (20.8%) had non-LBBB and QRSd <150 ms.

93 al deformation pattern is characteristic for LBBB and results from intraventricular dyssynchrony.

94 ents (group A; 27.4%) developed a persistent LBBB and the remaining 594 (group B; 72.6%) did not.

95 as an independent predictor in both RBBB and LBBB and, in addition, in LBBB, QRS/STT angle and ST J-p

96 tudy patients with left bundle branch block (LBBB) and 0, 1, 2, or >/=3 comorbidities, including rena

97 M-34 with CAD (20 normal activation [NA], 14 LBBB) and 25 without CAD (15 NA, 10 LBBB)-were studied.

98 Of these, 1175 had left bundle-branch block (LBBB) and 308 had non-LBBB.

99 erized by isolated left bundle branch block (LBBB) and a history of progressive left ventricular (LV)

100 ects of associated left bundle branch block (LBBB) and coronary artery disease (CAD) on peak cardiac

101 locity and pressure, with native conduction (LBBB) and during biventricular pacing at atrioventricula

102 etween that during left bundle branch block (LBBB) and LV pacing, reflects optimal resynchronization,

103 ween patients with left bundle-branch block (LBBB) and normal QRSd and if synchrony improved during p

104 mpact of new-onset left bundle branch block (LBBB) and permanent pacemaker implantation (PPI) after t

105 hree patients with DCM, 48 with CAD (16 with LBBB), and 25 without CAD (10 with LBBB) were studied.

106 al of 111 patients with DCM, 51 with CAD (29 LBBB), and 60 without CAD (30 LBBB) were studied with ec

107 hic morphology was left bundle branch block (LBBB), and in 15, it was nonspecific intraventricular co

108 Compared with women with no BBB, LBBB, and intraventricular conduction defect were strong

109 The t-IVT, CAD, LBBB, and QRS duration were univariate predictors of exe

110 pulse pressure) compared with atrial pacing-LBBB, and this improvement correlated with mechanical re

111 uggest that only a minority of patients with LBBB are ultimately diagnosed with acute myocardial infa

112 tion >/= 140 ms may warrant consideration in LBBB as an indication for further diagnostic evaluation

113 % ]; P=0.009) increase than BiV-Opt, against LBBB as reference; BiV-Opt and biventricular pacing at A

114 Persistent LBBB at hospital discharge was associated with a decreas

115 that patients with left bundle branch block (LBBB) be treated with cardiac resynchronization therapy

116 fect of evolving ST segment, T or Q waves or LBBB between serially obtained prehospital and hospital

117 In comparison with LBBB, biventricular pacing at separately preidentified h

118 Only among those with LBBB, both sexes had better survival with longer QRS dur

119 creased significantly in CRT-D patients with LBBB but not in non-LBBB patients.

120 V pacing reduced QRS duration by 21+/-10% in LBBB but only by 5+/-12% in LBBB+HF hearts.

121 ) in patients with left bundle-branch block (LBBB), but the clinical impact of this testing strategy

122 ated with better survival in both sexes with LBBB, but not in patients without LBBB.

123 reased with stress (NA by 4.7 +/- 2.7 l/min; LBBB by 4.0 +/- 2.3 l/min; all p < 0.001).

124 t-IVT became shortened (NA by 7 +/- 3 s/min; LBBB by 9 +/- 4 s/min) and correlated with a fall in the

125 raction </=35%, QRS duration >/=120 ms) with LBBB by ECG were prospectively included.

126 Left bundle branch block (LBBB) causes left ventricular (LV) dyssynchrony which is

127 h acute myocardial infarction, regardless of LBBB chronicity, and that a significant proportion of pa

128 n 22 dogs, 9 with acute LBBB, 7 with chronic LBBB combined with infarction (embolization; LBBB plus m

129 low direction in heart failure patients with LBBB compared to those without LBBB during early but not

130 tal activation time (LVTAT) in patients with LBBB compared with heterogeneous activation sequences an

131 Assessment of LBBB contraction pattern was superior to time-to-peak in

132 Two-thirds of patients (63%) had a typical LBBB contraction pattern.

133 Absence of a typical LBBB contraction was independently associated with incre

134 al 4-chamber view determined whether typical LBBB contraction was present.

135 ar, the effects of left bundle branch block (LBBB), coronary artery disease (CAD), and total isovolum

136 Most data pertain to LBBB delays.

137 ss I or II and ejection fraction </= 30% and LBBB derive substantial clinical benefit from CRT-D: a r

138 50.5 years of age on average at the time of LBBB diagnosis.

139 e achieved for patients with normal QRSd and LBBB during biventricular and LV pacing.

140 patients with LBBB compared to those without LBBB during early but not late diastole.

141 evelop a transient left bundle-branch block (LBBB) during exercise, but its prognostic significance i

142 Use of strict LBBB ECG criteria was not independently associated with

143 IAT during follow-up in 1,264 patients with LBBB enrolled in the MADIT-CRT (Multicenter Automatic De

144 in-hospital survival of 29,585 patients with LBBB enrolled in the National Registry of MI 2 June 1994

145 RT-D patients with left bundle branch block (LBBB) enrolled in MADIT-CRT (Multicenter Automatic Defib

146 erter defibrillator-CRT in patients with non-LBBB, especially when the QRS duration is <160 ms.

147 Patients with LBBB experienced a 25.3-mL/m(2) mean reduction in left v

148 stolic function in left bundle-branch block (LBBB)-failing hearts despite different electrical activa

149 and hemodynamics were obtained in dogs with LBBB-failing hearts during right atrial, LV, and BiV sti

150 prior conduction disturbances developed new LBBB following TAVI with a balloon-expandable valve, alt

151 d syndrome, including: 1) history of typical LBBB for >5 years; 2) LV ejection fraction (EF) >50%; 3)

152 with a suspected acute coronary syndrome and LBBB for urgent reperfusion therapy.

153 The latter 2 groups were defined as non-LBBB groups.

154 s patients with a QRSD 150 to 179 ms without LBBB had no improvement in survival with CRT-D, and thos

155 T-D, and those with a QRSD 150 to 179 ms and LBBB had only a modest improvement.

156 ors of all-cause mortality were TAVI-induced LBBB (hazard ratio [HR], 1.54; confidence interval [CI],

157 mong patients with left bundle branch block (LBBB) (hazard ratio [HR]: 0.58; p < 0.001) and no signif

158 t to patients with left bundle branch block (LBBB), heart failure patients with narrow QRS and nonspe

159 was significantly slower in LBBB+HF than in LBBB hearts (67+/-9 versus 44+/-16 ms, respectively), an

160 s optimal timing of LV stimulation in canine LBBB hearts.

161 ntional epicardial CRT in compromised canine LBBB hearts.

162 rker of diastolic mechanical dyssynchrony in LBBB hearts.

163 In LBBB and LBBB+HF animals, endocardial conduction was approximatel

164 l infarction than in eccentrically remodeled LBBB+HF hearts (19% versus 10%).

165 n by 21+/-10% in LBBB but only by 5+/-12% in LBBB+HF hearts.

166 pulse conduction was significantly slower in LBBB+HF than in LBBB hearts (67+/-9 versus 44+/-16 ms, r

167 hronic LBBB and heart failure (rapid pacing, LBBB+HF, and eccentric remodeling).

168 LBBB with tachypacing-induced heart failure (LBBB+HF, n=6).

169 dian difference in CURE-SVD (range, 0-1) for LBBB-HF group versus narrow-QRS-HF group (-0.40; 95% con

170 however, there was no sex difference in non-LBBB (HR: 0.95; 95% CI: 0.85 to 1.06; p = 0.37).

171 RT-D patients with left bundle branch block (LBBB) (HR: 0.51 [95% CI: 0.35 to 0.76], p < 0.001).

172 s effect on hearts with RBBB than those with LBBB (i.e., 5.5 +/- 1.1% vs. 29.5 +/- 5.0% increase in d

173 le, we describe the evolving epidemiology of LBBB in acute coronary syndromes and discuss controversi

174 Isolated LBBB in animals causes cardiac remodeling due to mechani

175 hanical dyssynchrony is induced by RBBB than LBBB in failing hearts, and the corresponding impact of

176 w-onset persistent left bundle branch block (LBBB) in patients undergoing transcatheter aortic valve

177 patients with non-left bundle branch block (LBBB; including right bundle branch block, intraventricu

178 However, the existence of LBBB-induced cardiomyopathy in humans remains uncertain.

179 rvations support the existence of a specific LBBB-induced cardiomyopathy resolved by CRT.

180 LBBB, intraventricular conduction defect, and RBBB combi

181 TAVI-induced LBBB is an independent predictor of mortality.

182 Additionally, longer QRS duration in LBBB is associated with better survival in both sexes.

183 In contrast, LBBB is most commonly caused by nonischemic pathologies.

184 ted with significantly larger scar size than LBBB is, and occlusion of a proximal LAD septal perforat

185 tients develop new left bundle-branch block (LBBB), its effect on clinical outcome is unclear.

186 , and dyslipidemia, and had more often a non-LBBB (left bundle branch block) wide QRS complex, and lo

187 py candidates with left bundle branch block (LBBB)-like electrocardiogram morphology (left ventricula

188 It is unclear whether new-onset LBBB may also impact the prognosis of patients after tra

189 d with resynchronization pacemakers, 13 with LBBB (mean QRS, 171 ms) and 9 with normal QRSd <120 ms (

190 investigate whether the absence of a typical LBBB mechanical activation pattern by 2DSE was associate

191 t arrhythmias with left bundle-branch block (LBBB) morphology.

192 Not all LBBB-morphology EIVA can be dismissed, and not all RBBB-

193 2.73 [95% CI, 1.78 to 4.13]; P < 0.001), but LBBB-morphology EIVA was not (hazard ratio, 0.82 [CI, 0.

194 Inclusion of patients with isolated LBBB-morphology EIVA, which often is idiopathic, may con

195 Patients with LBBB-morphology EIVAs had a mortality rate (2.5%) simila

196 o heart failure patients (narrow QRS [n=18], LBBB [n=11], NICD [n=23]) underwent 3-dimensional electr

197 ed dogs with acute left bundle-branch block (LBBB, n=10) and chronic LBBB with tachypacing-induced he

198 tion were compared among patients with RBBB, LBBB, nonspecific LV conduction delay, and QRS <120 ms.

199 ith STEMI or a new left branch bundle block (LBBB), of which 1,654 (60%) presented < or =12 hours.

200 all-cause 1-year mortality and (2) new-onset LBBB on the need for PPI at 1-year follow-up.

201 changes in ventricular activation induced by LBBB or CAD and is, by itself, a major determinant of pe

202 were acquired in heart failure patients with LBBB or matched patients without LBBB.

203 er reason, then categorized as having either LBBB or no LBBB and QRS duration of either 150 ms or gre

204 centers in Italy, we analyzed those without LBBB or pacemaker at admission (879 patients [82.9%]).

205 e the impact of (1) periprocedural new-onset LBBB or PPI post-TAVR on cardiac mortality and all-cause

206 ersistent ST-segment elevation as opposed to LBBB or Q waves.

207 mong patients with left bundle-branch block (LBBB) or longer QRS duration.

208 Patients without left bundle branch block (LBBB) or patients with smaller QRS duration (QRSd) respo

209 At rest, t-IVT was 8 s/min longer with LBBB (p < 0.001), was unaffected by CAD, and did not cor

210 h LBBB and 24.0% (n=107) in patients without LBBB (P=0.002).

211 ile range, 253-725) days in patients without LBBB (P=0.90).

212 ] index: 0.80 +/- 0.03 vs. 0.58 +/- 0.09 for LBBB, p < 0.04; CURE 0-->1 is dyssynchronous-->synchrono

213 ICD) were significantly (P < 0.001) lower in LBBB patients (0.47; P < 0.001) than in non-LBBB patient

214 LBBB patients (0.47; P < 0.001) than in non-LBBB patients (1.24; P = 0.257).

215 ection fraction was similar between RBBB and LBBB patients (24.9% vs. 25.0%; p = 0.98); however, RBBB

216 Sixty-three symptomatic LBBB patients (group A), 10 left ventricular ejection fr

217 < 0.001) and no significant effect among non-LBBB patients (HR: 1.05; p = 0.82, p for the difference

218 CRT-D was significantly increased among non-LBBB patients (HR: 3.62; p = 0.002, p for the difference

219 Non-LBBB patients (n=537; 30%) were divided into 2 groups ba

220 ystolic volume index (P<0.0001), whereas non-LBBB patients had smaller decreases (6.7 mL/m(2); P=0.18

221 rams from the LV free wall were later in the LBBB patients in absolute terms (155 ms [SD 23] versus 6

222 LBBB patients typically demonstrated (1) a single LV bre

223 -CRT study, the clinical benefit of CRT-D in LBBB patients was not attenuated by prior history of IAT

224 In non-LBBB patients with a normal PR interval, implantation of

225 In non-LBBB patients with a prolonged PR interval, CRT-D treatm

226 ta support the use of CRT-D in MADIT-CRT non-LBBB patients with a prolonged PR interval.

227 and MBF reserve is homogeneously reduced in LBBB patients with left ventricular systolic dysfunction

228 In non-LBBB patients with normal PR, CRT-D therapy was associat

229 eath was not significantly different between LBBB patients with or without history of IAT (HR: 0.50,

230 Long-term survival was better in LBBB patients with QRSd >/=150 ms (p = 0.02), but this d

231 ctor (P=0.006) of SPECT perfusion defects in LBBB patients without CAD.

232 ve reperfusion therapy (13.6% vs. 2.6%) than LBBB patients without chest pain; they were also more li

233 In contrast to LBBB patients, narrow QRS and NICD patients are characte

234 y increase subsequent arrhythmic risk in non-LBBB patients.

235 y in CRT-D patients with LBBB but not in non-LBBB patients.

236 tion fraction with CRT-D in LBBB than in non-LBBB patients.

237 vasodilator MCE and SPECT were undertaken in LBBB patients.

238 ger scar size than left bundle branch block (LBBB) patients do.

239 Among those with LBBB, patients with a QRSD >/=180 ms had a greater adjus

240 nefit was larger in concentrically remodeled LBBB plus myocardial infarction than in eccentrically re

241 LBBB combined with infarction (embolization; LBBB plus myocardial infarction, and concentric remodeli

242 all discriminate effectively between LLk and LBBB populations.

243 well they discriminated between the LLk and LBBB populations.

244 for studies reporting raw data on new-onset LBBB post-TAVR and the need for PPI or mortality at 1-ye

245 New-onset LBBB post-TAVR is a marker of an increased risk of cardi

246 New-onset LBBB post-TAVR was associated with a higher risk of PPI

247 Nearly half of MI patients with LBBB present without chest pain.

248 onary syndrome and left bundle branch block (LBBB) present a unique diagnostic and therapeutic challe

249 benefit was observed in patients with a non-LBBB QRS pattern (right bundle-branch block or intravent

250 r in both RBBB and LBBB and, in addition, in LBBB, QRS/STT angle and ST J-point depression in aVL wer

251 tion 26+/-7%) with left bundle-branch block (LBBB; QRS duration 174+/-18 ms) were atriobiventricularl

252 th a fall in the QRS duration (NA: r = 0.87; LBBB: r = 0.91), and CO increased with stress (NA by 4.7

253 m follow-up of MADIT-CRT study patients with LBBB randomized to CRT-D, there were differences in HF o

254 nto left, right, and indetermined-type BBBs (LBBB, RBBB, and intraventricular conduction defect, resp

255 In patients with LBBB receiving implantable cardioverter defibrillator-CR

256 RS duration and survival in patients without LBBB regardless of patient sex.

257 LBBB and QRS duration less than 150 ms or no LBBB regardless of QRS duration, was associated with low

258 n patients without left bundle branch block (LBBB) regardless of patient sex.

259 ere is no sex difference in patients without LBBB, regardless of QRS duration.

260 How LBBB-related effects on LV diastolic function may contri

261 r small differences in age, exercise-induced LBBB remained associated with a higher risk of primary e

262 chrony and impact of CRT in pure RBBB versus LBBB remains largely unknown.

263 hearts with acute left bundle branch block (LBBB) showed that endocardial left ventricular (LV) paci

264 inical composite score improved with CRT for LBBB subjects (odds ratio, 0.530; P=0.0034) but not for

265 odds ratio, 0.530; P=0.0034) but not for non-LBBB subjects (odds ratio, 0.724; P=0.21).

266 However in LBBB, systolic amplitude proved to be the only significa

267 increase in ejection fraction with CRT-D in LBBB than in non-LBBB patients.

268 after TAVI is higher in patients who develop LBBB than in patients who do not.

269 In those without LBBB, the mortality difference was modest and did not di

270 In patients with LBBB, there was a continuous relationship between broade

271 tion, (2) multiple left bundle-branch block (LBBB)-type VTs, and (3) an abnormal endocardial substrat

272 mmend that patients with new or presumed new LBBB undergo early reperfusion therapy, data suggest tha

273 ts with narrow QRS and NICD to patients with LBBB using high-density electroanatomic activation maps.

274 cardiac effects of left bundle-branch block (LBBB) using myocardial contrast echocardiography (MCE) t

275 ery wide QRSD with left bundle branch block (LBBB) versus those without LBBB.

276 In 52 of 66 LBBB VTs, the origin was from the RV perivalvular region

277 On the other hand, LBBB was associated with a higher short-term rate of pac

278 LBBB was more frequent after implantation of the Medtron

279 yses and inherent log-rank tests showed that LBBB was not associated with higher all-cause mortality,

280 New-onset LBBB was observed in 61 patients (30.2%) after TAVI, and

281 90% sensitivity and 82% specificity whether LBBB was present or not.

282 New-onset LBBB was the only factor associated with PPI following T

283 Left bundle branch block (LBBB) was present in 65 patients, right bundle branch bl

284 he mortality differences in patients without LBBB were attenuated in both sexes.

285 rom the cohort, 70 cases of exercise-induced LBBB were identified.

286 Patients with LBBB were less likely to experience at least one VT/VF e

287 Those with RBBB (compared with LBBB) were more likely to have ischemic heart disease (7

288 1 with CAD (29 LBBB), and 60 without CAD (30 LBBB) were studied with echocardiography and cardiopulmo

289 (16 with LBBB), and 25 without CAD (10 with LBBB) were studied.

290 [NA], 14 LBBB) and 25 without CAD (15 NA, 10 LBBB)-were studied.

291 tients with a QRSD >/=180 ms with or without LBBB, whereas patients with a QRSD 150 to 179 ms without

292 associated with a higher rate of persistent LBBB, which in turn determined higher risks for complete

293 proach among clinically stable patients with LBBB who do not have electrocardiographic findings highl

294 Patients with LBBB who experienced a first VTE had no change in the ri

295 y should be considered for all patients with LBBB who have symptoms consistent with MI.

296 Among patients with LBBB who received CRT-D, mortality is lower in women tha

297 bundle-branch block (LBBB, n=10) and chronic LBBB with tachypacing-induced heart failure (LBBB+HF, n=

298 independent predictor of incident HF only in LBBB, with more pronounced risk at QRS >/= 140 ms than a

299 mong patients with left bundle branch block (LBBB), women had a 21% lower mortality risk than men (HR

300 Among patients with LBBB, women receiving CRT-D had a lower relative death r