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1 A-Ag and the HRP labeled HA antibody (HRP-HA-Ab).
2 mily member lacking LTA-specific antibodies (Abs).
3 roduction was inhibited by a HLA-DR blocking Ab.
4 ut there were no differential effects on the AB.
5 and GAD65-Ab, and 1 had anti-Hu Ab and GAD65-Ab.
6 en patients positive for PLA2R1-Ab or THSD7A-Ab.
7 R9, which was not utilized in the absence of Ab.
8 tion on endothelial cells, or by an anti-VWF Ab.
9 tructure of F-actin complexed with the L253P ABD.
10 duces the immunopathological features of NOD ABD.
11 ive immune response by producing Ag-specific Abs.
12  we performed muscle cell coculture with the Abs.
13  verified by Western blotting using specific Abs.
14 validated by Western blotting using specific Abs.
15 rgence of ADAs in the case of anti-TNF-alpha Abs.
16 3-deficient mice using nondepleting anti-CD4 Abs.
17  (VGKCc) Ab, 2 (1.8%) had GAD65-Ab and VGKCc-Ab, 1 had TPO-Ab and GAD65-Ab, and 1 had anti-Hu Ab and
18 antibody and the antigen binding fragments F(ab')2 and Fab) targeting epidermal growth factor recepto
19 estricted specificity for protease-cleaved F(ab')2 fragments and did not bind the intact IgG counterp
20  A, diphenyleneiodonium, DNase or blocking F(ab')2 fragments to CD16, CD18, CD32 and CD64.
21 tor functions to proteolytically generated F(ab')2 fragments.
22 hereas reactivity against pepsin-generated F(ab')2 targets better discriminated between RA and HDs or
23          Reactivity against IdeS-generated F(ab')2 targets was found most frequently, whereas reactiv
24 alpha-radioimmunotherapy with (211)At-MX35-F(ab')2 was strong and dose-dependent.
25 ld-type mice injected with anti-mLAMalpha3 F(ab')2 were completely protected.
26 tage-gated potassium channel complex (VGKCc) Ab, 2 (1.8%) had GAD65-Ab and VGKCc-Ab, 1 had TPO-Ab and
27                           Anthon and Barret (AB) (2003) proposed modifications to this procedure.
28        Thus, the corresponding equation was: Abs=4.00(+/-0.16) [tannic acid]+0.17(+/-0.017).
29 valent immobilization of a coating antibody (Ab), a polyclonal anti-IgG, onto a treated PDMS surface.
30                 By using an anti-reticulon-1 Ab, a role for reticulon-1 in macrophage migration towar
31 n after excluding TPO-Ab and low-titer GAD65-Ab, Abs strongly suggesting an autoimmune cause of epile
32 atients may develop Abs also called antidrug Abs (ADA), directed against these anti-TNF-alpha Abs aft
33 quent nucleation of ABS or direct release of ABS aerosols.
34 (ADA), directed against these anti-TNF-alpha Abs after just a few weeks of treatment.
35                  We also prepared a specific Ab against the purified recombinant NC1 domain peptide.
36 onist-treated neonates could be blocked with Abs against CD80 and CD86.
37                          High titers of ADCC-Abs against H7N9 virus-infected cells were detected in s
38  (PD-L2) regulates the production of natural Abs against phosphorylcholine (PC).
39 d with alpha-gal or protein G to deplete IgG Ab. alpha-Gal-specific IgG1-4 Ab in individuals with and
40                                  Anti-TbKHC1 Ab also inhibited T. musculi growth, both in vitro and i
41 a major concern because patients may develop Abs also called antidrug Abs (ADA), directed against the
42  close to calculated interlayer distances in AB and ABC alpha-graphityne (3.255 and 3.206 A vs 3.266
43  (1.8%) had GAD65-Ab and VGKCc-Ab, 1 had TPO-Ab and GAD65-Ab, and 1 had anti-Hu Ab and GAD65-Ab.
44 1 had TPO-Ab and GAD65-Ab, and 1 had anti-Hu Ab and GAD65-Ab.
45                                    Anti-CD44 Ab and HA treatments reduced NF-kappaB translocation, IL
46                     Even after excluding TPO-Ab and low-titer GAD65-Ab, Abs strongly suggesting an au
47 ICAM-1 clustering in response to HLA class I Ab and prevents macrophage infiltration into cardiac all
48 annel complex (VGKCc) Ab, 2 (1.8%) had GAD65-Ab and VGKCc-Ab, 1 had TPO-Ab and GAD65-Ab, and 1 had an
49 ected in 7 patients (6.3%): 3 (2.7%) had TPO-Ab and voltage-gated potassium channel complex (VGKCc) A
50 nclusion, the prevalence of anti-C3b/anti-FB Abs and alternative pathway activation is similar in Ig-
51 ctivating B cells to proliferate and produce Abs and inflammatory cytokines.
52  and CD19(neg) ASCs secrete vaccine-specific Abs and show linked IGHV repertoires.
53       Treatment with anti-Marco-neutralizing Abs and the class A scavenger receptor antagonist polyin
54 ngiotensin II type 1 receptor antibody (AT1R-Ab) and anti-endothelial cell antibody (AECA).
55 space (where an interaction of the antibody (Ab) and the analog of E2 in the self-tuneable matrix tak
56 ylglutaryl-CoA reductase (HMGCR) antibodies (Abs), and the titer of these Abs is correlated with dise
57 AD65-Ab and VGKCc-Ab, 1 had TPO-Ab and GAD65-Ab, and 1 had anti-Hu Ab and GAD65-Ab.
58 , among the shortest known for this class of Abs, and achieved breadth with only 10% nucleotide somat
59 ated with stronger induction of neutralizing Abs, and more Ab-secreting cells in bone marrow.
60           The response of EF4.1 mice to an I-Ab-associated epitope of the F-MuLV envelope protein is
61 apeutic administration of anti-MSLN-blocking Abs attenuated BDL-induced fibrosis in WT mice.
62                                     Blockade Ab avidity to the GI.1 vaccine component peaked at day 3
63 h factor, and platelet-derived growth factor AB) before and after completing 2 therapy cycles.
64 o engineer allergen mutants with reduced IgE Ab binding for immunotherapy.
65  the cortisol in the sample, for the capture Ab binding sites.
66 teractions, hexamer formation, and CDC after Ab binds cell-surface Ag.
67                            We show that each ABS binds to a distinct place on F-actin.
68                                  The lineage Abs bore an anionic heavy chain complementarity-determin
69 he same human VH gene segment as an RV144 V2 Ab but paired with a mouse lambda L chain.
70 ancestor of a V2-glycan broadly neutralizing Ab, but this Ab type was not induced in the RV144 trial.
71 a IgG contains approximately 94% fucosylated Abs, but alloantibodies against, for example, Rhesus D (
72 development is that cross-reactive anti-DENV Abs can be protective or potentially increase disease vi
73 o neutralization, lysis of infected cells by Abs can effectively participate in HIV protection, as su
74                                              Abs can elicit a number of mechanisms to delete target c
75 8 heart recipients transplanted in Edmonton, AB, Canada, including 27 cases of AMR and 71 controls.
76 enerate long-lived plasma cells and generate Abs capable of neutralizing variant hemagglutinin-expres
77                       alpha-Gal-specific IgE Ab cause a special form of meat allergy characterized by
78 l subsets after activation with CD3 and CD28 Abs (CD3/28), transduction and culture alone, or after r
79     Notably, in vitro phosphorylation of the ABD closely recapitulated the previously described in vi
80 uptake and presentation of in vivo-formed Ag-Ab complexes by i.v. injecting mice with Ag-specific Abs
81 sociation of the K409R parental Ab into half-Ab controls the rate of the reaction.
82 demic regions and cross-reactive antibodies (Abs) could potentially affect ZIKV pathogenesis.
83                  Combined TIRAP and anti-LTA Ab deficiencies underlie staphylococcal disease in this
84 exosomes were significantly inhibited within Ab-deficient DHLMP2A mice compared with wild-type mice.
85 n that complement-dependent cytotoxicity and Ab-dependent cellular cytotoxicity favored a membrane-pr
86 including complement-dependent cytotoxicity, Ab-dependent cellular cytotoxicity, and Ab-dependent cel
87 er MAbs specific for V3, CD4bs, and gp41 for Ab-dependent cellular phagocytosis (ADCP) activity, impl
88 favored a membrane-proximal epitope, whereas Ab-dependent cellular phagocytosis favored an epitope po
89 ity, Ab-dependent cellular cytotoxicity, and Ab-dependent cellular phagocytosis.
90 otective or potentially increase disease via Ab-dependent enhancement.
91 that the staining intensity by anti-Valpha24 Abs depends on whether Ser or Arg is encoded.
92                            We show that EGFR Abs directed against clinically relevant epitopes can be
93                    The detection of anti-PEG abs directly from diluted blood serum was achieved withi
94                     In addition, islets from Ab+ donors were larger and contained a higher number of
95 les containing proinsulin in beta-cells from Ab+ donors, suggesting a defect in proinsulin conversion
96 nduced TNFR family-related protein agonistic Ab DTA-1 (rat or murinized IgG2a) can induce the develop
97 distribution of the immunospecific complexes Ab(E2)x (x=0, 1, 2) dependent on E2 concentration.
98         Together with previous base editors, ABEs enable the direct, programmable introduction of all
99 Ab lineage targeting the Env V2 apex and the Ab-Env co-evolution that led to development of neutraliz
100 t of platelet-derived growth factor-AB (PDGF-AB) expedites migration through native tissue.
101 connections between IG genomic diversity and Ab-expressed repertoires and structure.
102 exes by i.v. injecting mice with Ag-specific Abs followed by the cognate Ag.
103 ype are critical to generate ideal surrogate Abs for testing immuno-oncology mechanisms in vivo.
104 of risks in patients receiving IL-1 blocking Abs for treatment of chronic inflammatory disorders.
105                                  Traditional Ab formats (IgG, IgM) are expensive and laborious to pro
106                                        NMDAR-Ab from patients and healthy subjects do not compete for
107                         We unveil that NMDAR-Ab from psychotic patients alter NMDAR synaptic transmis
108       To test the functional consequences of Ab fucosylation, we produced V-gene-matched recombinant
109  patients with a positive ECXM and with AT1R-Ab >17 U/ml, although this did not reach statistical sig
110                                    Anti-CD20 Ab had a major effect on alloreactive CD4(+) T cells, in
111 fers enhanced FcRn interaction in vitro, and Abs harboring either the Q311R or TLQ mutations have ser
112                                    Anti-CD20 Ab has recently been used to prevent alloimmunization.
113 ction of new rAb fragments and single-domain Abs have reinvigorated the concept of passive immunizati
114 oded and somatically mutated residues on the Ab heavy chain.
115 lpha antibody and horseradish peroxidase (MP-Ab-HRP) were used to efficiently capture ERalpha from th
116 at is altered upon binding of PSMA with PSMA-Ab immobilized on N-AuNPs/ITO.
117 osing because of an anti-idiotypic anti-drug Ab immune response.
118 to deplete IgG Ab. alpha-Gal-specific IgG1-4 Ab in individuals with and without meat allergy were ass
119 , a possible role for the BCR or circulating Ab in mediating CTL responses to B cell-derived exosomes
120 d specific measurement of circulating THSD7A-Ab in patients with MN.
121 , highlighting the interest in inducing such Abs in future HIV vaccine trials.
122 mmune activation is often explored using rat Abs in immunocompetent mouse models.
123 le, when type I IFN signaling was blocked by Abs in Rag1(-/-) mice, the mice showed dramatic weight l
124        To further understand the role of the Abs in the pathology, we performed muscle cell coculture
125 ined by using the square root of absorbance (Abs) in order to adjust the residues of the curve.
126  we compared ADCC-mediating antibodies (ADCC-Abs) in sera collected from healthy infants, children an
127 ting a role for the BCR, but not circulating Ab, in DC-derived exosome responses.
128 2 is internalized after ligation by specific Abs, including H4B4, and traffics rapidly but transientl
129                                         Exon Ab inclusion correlated with predicted free energies of
130 a measure of the breadth of FcgammaR-binding Abs induced by the RV144 trial.
131                                    We employ ab initio and density functional methods to investigate
132        Here we develop an informatics-guided ab initio approach to accelerate the design and discover
133                  We demonstrate that a fully ab initio approach, relying on the strongly constrained
134                                Combined with ab initio calculation, our results show that TaIrTe4 is
135    This destabilizing effect is supported by ab initio calculations and experimentally by a normalize
136  that accounts for uncertainties not only in ab initio calculations and thermodynamic models but also
137   However, poor scaling often renders direct ab initio calculations intractable for large or complex
138                                          Our ab initio calculations on gold nucleation suggest that t
139                                              Ab initio calculations reveal that the primary assumptio
140                                              Ab initio calculations suggest that carbon-fluorine bond
141 lution of the diffusion equation, as well as ab initio calculations, support our interpretation.
142 copies, neutron scattering, calorimetry, and ab initio calculations.
143 racterized by photoelectron spectroscopy and ab initio calculations.
144 r supported by IR-MS action spectroscopy and ab initio calculations.
145                              Comparison with ab initio computations supports the assignment of the cl
146 ed from the protein structure database or by ab initio docking of monomer structures resolved by expe
147                                              Ab initio electronic structure calculations reveal that
148                       We map out an accurate ab initio ground-state potential energy surface of the K
149 ductivity kappa of MgSiO3 perovskite (pv) by ab initio lattice dynamics calculations combined with ex
150            Following the general paradigm of ab initio methods for low-resolution restoration of solu
151                                       Modern ab initio methods have rapidly increased our understandi
152 ettaAntibody, one of the leading H3-specific ab initio methods, both in accuracy and speed.
153              However, a genuinely predictive ab initio model of water has eluded scientists.
154                                              Ab initio molecular dynamics (AIMD) simulations and mole
155 OO(-))(H2SO4), and has also been verified by ab initio molecular dynamics (AIMD) simulations.
156                                              Ab initio molecular dynamics (AIMD)-informed EXAFS analy
157                                              Ab initio molecular dynamics simulations have confirmed
158 am, scanning tunneling microscopy (STM), and ab initio molecular dynamics.
159        Here we employ a combined approach of ab initio protein modelling and (13)C-direct detection N
160            FTIR/smog chamber experiments and ab initio quantum calculations were performed to investi
161 X-ray crystallography, NMR spectroscopy, and ab initio quantum-mechanical calculations.
162 esent a novel method, Sphinx, which combines ab initio techniques with the potential extra structural
163 ion-containing systems at the semiempirical, ab initio, and density functional levels of theory are r
164                           Here, we report an ab-initio based computational study of thermal transport
165  using the Boltzmann transport framework and ab-initio calculations.
166 ems by means of continuum, tight-binding and ab-initio calculations.
167                              Here we develop ab-initio theoretical method for correlated electron mat
168       The observations are in agreement with ab-initio-based calculations, which reveal a very narrow
169 , and risk factors for failure of standalone ab interno gelatin microstent implantation with mitomyci
170  trainees performed nine trabectome-mediated ab interno trabeculectomies in pig eyes (n = 63).
171                                              Ab intio molecular dynamics simulations show that the el
172 ns, where dissociation of the K409R parental Ab into half-Ab controls the rate of the reaction.
173  their physiologic target Ag, anti-chromatin Ab, into mice lacking Fcer1g, Myd88, or both and studied
174 CR) antibodies (Abs), and the titer of these Abs is correlated with disease activity.
175 fically its C-terminal actin-binding domain (ABD), is a target of PKA.
176 g a B-1a phenotype, and produced PC-reactive Abs largely of the T15 Id.
177 oral effects, serum IL-4, IgE, and anti-drug Ab levels.
178 A did not directly target B cells to elevate Ab levels.
179 ted with a difference in serum AMA1-specific Ab levels.
180 or PS and photothermal therapy of anti-CD11b Abs-linked gold nanorods (GNRs-CD11b) are combined to tr
181  variants that escape cellular and antibody (Ab)-mediated immune pressure, yet possess contemporary f
182                                              Ab-mediated enhancement of IFN production required Fc ga
183 hat complements the mucous and secretory IgA Ab-mediated system in the protection of intestinal mucos
184 is underlying differences between the SW and AB methods for indirect assessment of onion flavor and n
185                For example, although natural Abs (NA) and complement are key components of the innate
186 t MOG-ab NMOSD and 87.5% in the heterogenous ab-negative NMOSD cohort.
187 .1% against AQP4-ab NMOSD, 95.2% against MOG-ab NMOSD and 87.5% in the heterogenous ab-negative NMOSD
188 and with a specificity of 87.1% against AQP4-ab NMOSD, 95.2% against MOG-ab NMOSD and 87.5% in the he
189 uced V-gene-matched recombinant anti-RhD IgG Abs of the four different subclasses (IgG1-4) with and w
190  L253P mutation in the actin-binding domain (ABD) of beta-III-spectrin causes high-affinity actin bin
191 mice with the chimerized and murinized DTA-1 Abs on a C57BL/6-matched IgG2c isotype resulted in reduc
192                               Recognition of Ab-opsonized pathogens by immune cells triggers both TLR
193 th was prevented with neutralizing anti-IL-2 Ab or STAT5/JAK3 inhibitors, indicating that STAT5 signa
194 differs between patients positive for PLA2R1-Ab or THSD7A-Ab.
195  volatilization and subsequent nucleation of ABS or direct release of ABS aerosols.
196 ne responses can be manipulated in directing Abs or T cells based on how efficiently the targeted Ag
197 ifficult to measure the titer of Ag-specific Abs or the expansion of Ag-specific B cells after immuni
198 a gradient of platelet-derived growth factor-AB (PDGF-AB) expedites migration through native tissue.
199 phase has zero area thermal expansion in the ab-plane.
200 vial tissues from anti-citrullinated protein Ab-positive arthralgia, undifferentiated arthritis (UA),
201                     The prevalence of THSD7A-Ab-positive patients is unknown, and it is unclear wheth
202 DR-AB rates, greater compliance with the MDR-AB prevention bundle did not lead to reductions in MDR-A
203                                  Antibodies (Abs) produced by immunoglobulin (IG) genes are the most
204  we studied B cell cytokine secretion and/or Ab production across obesity models.
205 rived hematopoiesis, responsible for natural Ab production and rapid immune responses.
206 uction by T cells and thereby limits natural Ab production by B-1 cells.
207 T follicular helper numbers and the abnormal Ab production previously reported in these mice.
208 significantly enhances otherwise compromised Ab production to inactivated influenza vaccine.
209 nced GC B cell responses and anti-Plasmodium Ab production.
210 ted in increased germinal center B cells and Ab production.
211 or integrating IG genotyping with functional Ab profiling data as a means to better predict and optim
212         Importantly, a monoclonal anti-TIM-4 Ab promoted allograft tolerance, and this was dependent
213 the presence of antibody-functionalized QDs (Ab-QDs) onto the test line.
214 bodies: the visual immunogold OnSite Leprosy Ab Rapid test [Gold-LFA] and the quantitative, luminesce
215 acteristics associated with decreases in MDR-AB rates, greater compliance with the MDR-AB prevention
216 ion bundle did not lead to reductions in MDR-AB rates.
217       A total of 30 bogs were sampled in the ABS region, and 5 were sampled in central Alberta.
218 e envisage the ICA as being a useful tool in Ab repertoire analysis for future clinical trials.
219 ) titers are associated with protection, the Ab repertoire induced by LATVs remain uncharacterized.
220 g error-prone immunosequencing datasets into Ab repertoires is a fundamental problem in immunogenomic
221 with various H1N1 or H3N2 strains, the acute Ab response against HA exhibited an inherent bias toward
222 influenza viruses elicited an Igkappa-biased Ab response directed against the HA globular head and st
223                        In these mice, the V2 Ab response was focused on a linear epitope that did not
224 /2 In a cynomolgus model of T cell-dependent Ab response, the CTLA4-Ig variant MEDI5265 could be form
225 , the mechanisms by which targeting improves Ab responses are poorly understood.
226 tion with an MVA vector elicits cellular and Ab responses in the FRT.
227 city as assessed by their ability to inhibit Ab responses in vitro.
228            We hypothesized that HIV-specific Ab responses regulate late IFN production.
229 e of CX3CR1-mediated sampling did not affect Ab responses to a noninvasive S.
230              In addition, Chlamydia-specific Ab responses were dysregulated in CCR7-deficient mice, d
231                            Allergen-specific Ab responses were subsequently boosted with molecules co
232 lowed the induction of allergen-specific IgE Ab responses without allergen-specific T cell help.
233  with flagellin did not affect SseB-specific Ab responses, it modestly boosted CD4 responses.
234 , plasma cell generation, and virus-specific Ab responses.
235 iding new tools to study germinal center and Ab responses.
236               Immunoprecipitation with Sesn2 Ab revealed that cardiac Sesn2 forms a complex with AMPK
237 ructural characterization of one of these V2 Abs revealed how the linear V2 epitope could be engaged,
238 lization for the asymmetric assembly of the "AB" ring and the construction of the all-carbon quaterna
239 pared with the effect of a control anti-CD20 Ab (rituximab) at concentrations that triggered similar
240 nger induction of neutralizing Abs, and more Ab-secreting cells in bone marrow.
241 ciated with low frequencies of ZIKV-specific Ab-secreting cells in lymph nodes and bone marrow, corre
242  provide help to B cells for developing into Ab-secreting cells, were similar between responders and
243 production of Ag-specific memory B cells and Ab-secreting plasma cells.
244        The electrochemical, aptamer-based (E-AB) sensor platform provides a modular approach to the c
245 is improvement comes the ability to deploy E-AB sensors directly in situ in the veins of live animals
246                 Specifically, we have used E-AB sensors to perform the multihour, real-time measureme
247 a general approach to compatibly interface E-AB sensors with complex biological samples.
248             Electrochemical aptamer-based (E-AB) sensors offer advantageous analytical detection abil
249         Pyruvate determinations according to AB significantly reduced absorbance interference from co
250 captured by antibody-decorated silica beads (Ab-SiO2) onto the conjugate pad and the sample flows by
251  pathway, we conjugated SOD with antibodies (Ab/SOD, size 10nm) to plasmalemmal vesicle-associated p
252              IgG from patients with anti-C3b Abs stabilized C3bBb and perturbed C3b binding to comple
253 were significantly increased in the rumen of AB steers.
254 ter excluding TPO-Ab and low-titer GAD65-Ab, Abs strongly suggesting an autoimmune cause of epilepsy
255                                              Ab structure prediction has made great strides, but accu
256                                        Serum Abs suggesting a potential autoimmune etiology were dete
257  a significant minority had detectable serum Abs suggesting an autoimmune etiology.
258 nt an immune response against anti-TNF-alpha Abs, suggesting that immune complexes are a major determ
259 nd they were not blocked by anti-HLA class I Abs, suggesting that KIR2DL3, in addition to the traditi
260 orescent treponemal antibody absorption [FTA-ABS] test, and Inno-LIA line immunoassay).
261  aim was to test the potential in mice of an Ab that blocks the proteolytic capacity of uPA in the CI
262 ith delayed meat allergy display IgE and IgG Ab that selectively recognize the alpha-gal epitope on B
263 ch as the injection of integrin-neutralizing Abs that inhibit the entry of circulating lymphocytes in
264 reased transmission risk was correlated with Abs that reacted with a linear epitope at a lysine resid
265        We engineered trispecific antibodies (Abs) that allow a single molecule to interact with three
266   People exposed to DENV develop antibodies (Abs) that strongly neutralize the serotype responsible f
267                                  Trispecific Abs thus constitute a platform to engage multiple therap
268 s, failure to establish long-term protective Ab titers against S. aureus was not a consequence of dim
269 m and monophosphoryl lipid A (MPL), blockade Ab titers peaked early, with no increase in titer follow
270  nodes and bone marrow, correlating with low Ab titers.
271  proteins, and correlated strongly with ADCC-Abs titers against H7N9 virus-infected cells.
272                                         ADCC-Abs titers directed against H7N9 HA or NA proteins.
273            Further analysis showed that ADCC-Abs titers were significantly higher toward H7N9 NP, as
274 w glycan-specific maturation enables a human Ab to cope with pathogen escape mechanisms and will aid
275                                 Neutralizing Ab to IFN-gamma, but not IL-17, inhibited nevus developm
276 r catalyzing the transfer hydrogenation from AB to nitro compounds, leading to the green synthesis of
277 sis and ocular pathology, yet the ability of Ab to protect against HSV-1 is deemed limited due to the
278 imultaneously measure FcgammaR dimer-binding Abs to 32 different HIV Ags, providing a measure of the
279 e subdominant V2-glycan broadly neutralizing Abs to develop.
280                               Targeting ADCC-Abs to internal proteins may be a potential mechanism of
281 munized mice; thus, vaccination can generate Abs to regions that are not targeted in natural infectio
282 ion of HIV-1 infection risk with antibodies (Abs) to the V1V2 region of gp120 with high antibody-depe
283 while simultaneously inducing high levels of Abs toward hemagglutinin.
284 infection and to determine whether anti-PD-1 Ab treatment improves fungal clearance.
285 In this study, anti-sclerostin antibody (Scl-Ab) treatment effectively improved bone mass and dramati
286  V2-glycan broadly neutralizing Ab, but this Ab type was not induced in the RV144 trial.
287 (MR) imaging, and biannual automated breast (AB) ultrasonography (US) in women with BRCA1 and BRCA2 m
288 e produce significantly less Ag-specific IgM Abs upon immunization with T cell-independent Ags, and t
289                                              AB US did not depict additional cancers.
290                                   Conclusion AB US may not be of added value to yearly FFD mammograph
291      This variable domain of an H chain-only Ab (VHH or nanobody) significantly inhibited both phosph
292                                  More than 1 Ab was detected in 7 patients (6.3%): 3 (2.7%) had TPO-A
293                                           V2 Abs were isolated that used the same human VH gene segme
294       CD13 and VLA-4 blocking and activating Abs were used in flow-based adhesion assays, live-cell i
295 idrug-resistant Acinetobacter baumannii (MDR-AB) were assessed.
296 erence may explain the enhanced induction of Abs when targeting Ag to cDC1 using hXCL1 and hXCL2, and
297 fector cells via low-affinity anti-human IgE Abs with dissociation constants in the 10(-6) to 10(-8)
298 ized with SERCA2a 971-990 showed Ag-specific Abs with enhanced production of IgG2a and IgG2b isotypes
299  the hydrolysis of ammonia borane (H3 NBH3 , AB) with a turnover frequency (TOF) of 4896.8 h(-1) and
300 mice, which lack secreted and membrane-bound Ab, yet harbor marginal zone and follicular B cells.

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